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AHSA1 — TGFB1

Text-mined interactions from Literome

Liao et al., Cell Res 2001 (MAP Kinase Signaling System) : The data indicate that the activation of p38 is involved in TGF-beta1 mediated gene expression and apoptosis
Sato et al., J Invest Dermatol 2002 (Scleroderma, Systemic) : Furthermore, a specific inhibitor of p38 suppressed transforming growth factor beta stimulation of collagen type I mRNA and the alpha2 ( I ) collagen promoter activity
Frippiat et al., Free Radic Biol Med 2002 : Here we show that sustained p38 ( MAPK ) phosphorylation is responsible for both H2O2 induced overexpression of TGF-beta 1 and subsequent TGF-beta 1-induced appearance of these biomarkers
Dai et al., Am J Respir Cell Mol Biol 2003 (Fibrosis) : With diesel exhaust particles, tetramethylthiourea and deferoxamine had no effect, but extracellular signal regulated kinase and p38 inhibitors completely blocked effects on procollagen and TGF-beta 1
Ungefroren et al., J Biol Chem 2005 : We have recently shown that induction of biglycan (BGN) expression by transforming growth factor-beta1 ( TGF-beta1 ) required sequential activation of both Smad and p38 mitogen activated protein kinase signaling ( Ungefroren, H., Lenschow, W., Chen, W.-B., and Kalthoff, H. ( 2003 ) J. Biol. Chem. 278, 11041-11049 )
Guo et al., World J Gastroenterol 2005 : Since p38 phosphorylation induced by TGF-beta1 plays an important role in caspase activation and apoptosis, TPA and EGF induced p38 phosphorylation may not be requisite for their anti-apoptotic function
Perng et al., Chest 2007 (Asthma...) : Pretreatment with dexamethasone inhibited TGF-beta1 production at both messenger RNA and protein levels by inhibiting p38 MAP kinase phosphorylation
Ding et al., J Biol Chem 2008 (Disease Models, Animal...) : FRNK overexpression blocks TGF-beta1 induced ERK or p38 MAPK activation in the presence, and surprisingly, in the absence of FAK
Zimpelmann et al., American journal of physiology. Renal physiology 2009 (MAP Kinase Signaling System) : Stimulation of p38 MAPK phosphorylation by Ang- ( 1-7 ) leads to release of arachidonic acid and production of TGF-beta1 and extracellular matrix proteins
Houde et al., J Biol Chem 2009 : We show here that early events consisted of a trend toward increased expression of extracellular signal regulated kinase ( ERK ), and then TGF-beta1 significantly induced the activation of p38 and Smad2 in a time dependent manner
Wei et al., Cell Biol Int 2013 : Transforming growth factor-beta1 ( TGF-beta1 ) is the key inducer of EMT and the p38 mitogen activated protein kinases ( p38 MAPK ), an major intracellular signal transduction pathway is involved in TGF-beta1 induced EMT