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ATF2 — EPHB2
Text-mined interactions from Literome
Lewis et al., Oncol Res 2005
(Breast Neoplasms) :
In contrast, the
MEK-ERK1/2 inhibitor, PD98059, or the JNK inhibitor, SP600125,
had no significant effect on DNA binding of
ATF-2
Baan et al., Mol Endocrinol 2006
:
Because JNK is also capable of phosphorylating ATF2, we assessed the
involvement of JNK,
ERK1/2 and p38 in the insulin induced two-step
ATF2 phosphorylation in JNK expressing A14 fibroblasts and 3T3L1-adipocytes ... We conclude that sequential activation and nuclear appearance of
ERK1/2 and JNK, rather than p38,
underlies the two-step insulin induced
ATF2 phosphorylation in JNK expressing cells
Liu et al., Arch Toxicol 2013
:
Taken together, these data indicated that the increasing of ATF2 expression is mediated via oxidative stress induced by arsenic in SV-HUC-1 cells, and JNK or p38 rather than
ERK is
responsible for arsenic induced
ATF2 expression