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ANGPT2 — CALM3
Text-mined interactions from Literome
Yang et al., Atherosclerosis 2008
:
Insulin plus
Ang II inhibited PP-2A activity by 57+/-7 % ( P < 0.05 ) and
stimulated autonomous
CaM kinase II activity by 120+/-14 % ( P < 0.05 ), both by an apocynin-sensitive pathway ... It is concluded that insulin in the presence of NO stimulates cGMP which stimulates PP-2A activity causing inhibition of autonomous CaM kinase II activity and thus VSMC migration, and that insulin in the
presence of
Ang II inhibits PP-2A and stimulates autonomous
CaM kinase II activities by a NAD ( P ) H oxidase dependent mechanism which are associated with insulin stimulated NAD ( P ) H oxidase dependent migration
Eguchi et al., J Biol Chem 1996
:
The
calmodulin inhibitor, calmidazolium, and the tyrosine kinase inhibitor, genistein, completely
blocked MAPK activation by
Ang II as well as by the Ca2+ ionophore A23187
Graves et al., J Biol Chem 1997
:
The activation of p70 ( S6K ) by calcium stimuli or
Ang II was
independent of
calmodulin but correlated well with the activation of the recently identified, nonreceptor calcium dependent tyrosine kinase ( CADTK ) /PYK-2