Gene interactions and pathways from curated databases and text-mining

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ANGPT2 — CALM3

Text-mined interactions from Literome

Yang et al., Atherosclerosis 2008 : Insulin plus Ang II inhibited PP-2A activity by 57+/-7 % ( P < 0.05 ) and stimulated autonomous CaM kinase II activity by 120+/-14 % ( P < 0.05 ), both by an apocynin-sensitive pathway ... It is concluded that insulin in the presence of NO stimulates cGMP which stimulates PP-2A activity causing inhibition of autonomous CaM kinase II activity and thus VSMC migration, and that insulin in the presence of Ang II inhibits PP-2A and stimulates autonomous CaM kinase II activities by a NAD ( P ) H oxidase dependent mechanism which are associated with insulin stimulated NAD ( P ) H oxidase dependent migration
Eguchi et al., J Biol Chem 1996 : The calmodulin inhibitor, calmidazolium, and the tyrosine kinase inhibitor, genistein, completely blocked MAPK activation by Ang II as well as by the Ca2+ ionophore A23187
Graves et al., J Biol Chem 1997 : The activation of p70 ( S6K ) by calcium stimuli or Ang II was independent of calmodulin but correlated well with the activation of the recently identified, nonreceptor calcium dependent tyrosine kinase ( CADTK ) /PYK-2