◀ Back to IL1B

CALM3 — IL1B

Text-mined interactions from Literome

Rogers et al., J Infect Dis 1999 : Thus, amphotericin B acts as an ionophore to increase [ Ca++]i and activates calmodulin mediated expression of IL-1beta in human monocytes
Asai et al., FEMS Immunol Med Microbiol 2000 (Shock, Septic) : A CaM kinase activator reduced the induction of IL-1beta in the serum of mice when administered with compound 506, and protected the mice against the lethal toxicity
Simiantonaki et al., Exp Mol Pathol 2002 (Breast Neoplasms...) : IL-1beta stimulation had no effect on endothelial CAM expression
Wang et al., Clin Exp Rheumatol 2002 (Osteoarthritis) : IL-1 beta decreased the accumulation of aggrecan, hyaluronan and type II collagen in the CAM and increased intracellular MMP-1, -3 and -13 at a concentration of 100 pg/ml. Xylosan polysulphate and chrondroitin polysulphate restored the expression of these CAM molecules in these IL-1 beta treated cultures
Ikuta et al., Ryumachi 1992 (Arthritis, Rheumatoid) : In the intracellular signal transduction mechanism for the production of IL-1 beta by these cells following IFN-gamma stimulation, protein kinase C and calmodulin may be involved as second messengers
Nakano et al., Immunobiology 1993 : Though the CaM inhibitor inhibits the mRNA expression and the active molecule production of IL-1 beta , it does not affect those of TNF-alpha
Jiang et al., Neurochem Int 1997 : Since adhesion molecules ( AM ) induced on endothelial cells ( EC ) play an important role in T-cell migration into the CNS, the objective of this study was to examine the effect of IFN beta-1b on the expression of the AM, ICAM-1, V-CAM and E-selection induced on EC by IFN-gamma, TNF-alpha, or IL-1 beta