◀ Back to CDK2

CDK18 — CDK2

Text-mined interactions from Literome

Chae et al., Laryngoscope 2000 (Cholesteatoma, Middle Ear) : Cyclin-CDK complexes are in turn regulated by the cyclin dependent kinase inhibitors ( CDKIs ), which generally inhibit cell cycle progression
Okahashi et al., J Cell Biochem 2001 : In this study, we found that stimulation of M-BMMPhis with sODF/RANKL induced a transient expression of cyclin dependent kinase inhibitors ( CDK inhibitors ) p21 ( WAF1/CIP1 ) and p27 ( KIP1 ) by 24 h
You et al., Genes Dev 2002 : CDK2 activity is regulated by phosphorylation/dephosphorylation, subcellular localization, cyclin levels, and cyclin dependent kinase inhibitors ( CKIs )
Eluère et al., J Cell Sci 2007 : Orderly progression through the eukaryotic cell cycle is a complex process involving both regulation of cyclin dependent kinase activity and control of specific substrate-Cdk interactions
Lee et al., Carcinogenesis 2007 : The protein and mRNA levels of CDK inhibitors ( cyclin dependent kinase inhibitors ), such as p21 ( CIP1/WAF1 ) and p27 ( KIP1 ), were gradually increased after acteoside treatment in a time dependent manner
Kwon et al., Cancer Lett 2010 (Adenocarcinoma...) : Induction of G1 arrest by widdrol was correlated with induction of Chk2, p53 phosphorylation and CDK inhibitor p21 expression as well as inhibition of cyclin E, cyclin dependent kinase ( CDK2 ) and retinoblastoma protein ( pRB )
Lee et al., Exp Mol Med 2009 (Neoplasms) : Tight CDK regulation involves cyclin dependent kinase inhibitors ( CKIs ) which ensure the correct timing of CDK activation in different phases of the cell cycle
Molenaar et al., Genes Chromosomes Cancer 2012 (Chromosome Aberrations...) : The activity of the cyclin-CDK complexes is regulated by cyclin dependent kinase inhibitors ( CDKIs ) and multiple signal transduction routes that converge on the cell cycle
Mandal et al., Oncogene 1998 : Interferon-induces expression of cyclin dependent kinase-inhibitors p21WAF1 and p27Kip1 that prevent activation of cyclin dependent kinase by CDK activating kinase (CAK)