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CCL2 — CTR9

Text-mined interactions from Literome

Ahamed et al., J Biol Chem 2002 (MAP Kinase Signaling System) : In contrast, PAF induced CCL2 production was greatly reduced in mPAFR cells ... Pertussis toxin and U0126, which inhibit G ( i ) and p44/42 mitogen activated protein kinase ( ERK ) activation, respectively, caused very little inhibition of PAF induced CCL2 production ( approximately 20 % inhibition ) ... Whereas PAF induced CCL2 production is, in large part, mediated independently of G ( i ) activation or ERK phosphorylation, LTC ( 4 ) generation requires ERK phosphorylation, which is mediated by different G proteins depending on the phosphorylation status of the receptor
Beaudeux et al., Clin Chim Acta 2004 (Inflammation) : The effects of PAF and oxidised LDL on the production of monocyte chemoattractant protein-1 and interleukin-8 were also examined
Venkatesha et al., J Biol Chem 2004 : Interestingly, PAF induced significantly higher NF-kappaB and nuclear factor of activated T cells ( NFAT ) -luciferase activity as well as CCL2 production in cells expressing DeltaST-PAFR than those expressing PAFR ... Furthermore, a Ca2+/calcineurin inhibitor completely inhibited PAF induced NFAT activation and CCL2 production but not NF-kappaB activation
Verouti et al., Atherosclerosis 2011 : PAF effects on MCP-1 and IL-6 secretion in U-937 monocytes in comparison with oxLDL and IL-1ß effects
Jocks et al., Nephrol Dial Transplant 1998 (Glomerulonephritis) : These studies were designed to determine the possible role of platelet activating factor (PAF) in the production of monocyte chemoattractant protein-1 ( MCP-1 ) in glomerular immune injury ... These data show that PAF , derived from glomerular resident cells following antibody and complement induced injury, stimulates MCP-1 expression