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CAPN8 — DES

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Blunt et al., Am J Physiol Heart Circ Physiol 2007 (Reperfusion Injury) : We next determined that ventricular myocytes that underwent Ca ( 2+ ) overload also demonstrated a calpain dependent disruption of desmin that could be reduced by H ( 2 ) O ( 2 ) /p38 MAPK activation ... In a final series of experiments, we demonstrated that cardiac myofilaments exposed to recombinant phosphorylated HSP27, but not nonphosphorylated HSP27, had a significant reduction in the calpain induced degradation of desmin compared with non-HSP27 treated myofilaments
Chang et al., J Sci Food Agric 2012 (Postmortem Changes) : Therefore, our results suggest that BM µ-calpain with a faster and more extensive activation and autolysis would play a relatively dominant role in dictating degradation of desmin and troponin-T in postmortem duck muscle