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EGR1 — RNF19A

Text-mined interactions from Literome

Rolli et al., J Biol Chem 1999 : We conclude that egr-1 induction by anisomycin is mediated by p38/SAPK2 and probably by MSK1
Wu et al., Blood 2002 (MAP Kinase Signaling System) : Thrombin mediated induction of Egr-1 was blocked by inhibitors of MEK1/2, but not by inhibitors of protein kinase C, phosphatidylinositol 3-kinase, or p38 mitogen activated protein kinase ( MAPK )
Schaefer et al., Cell Signal 2004 (Leukemia, Erythroblastic, Acute...) : The p38 inhibitor SB 203580 enhanced ERK1/2 phosphorylation and egr-1 mRNA levels
Gorina et al., Glia 2011 : p38 exerted a strong influence on LPS induced gene expression by regulating the phosphorylation of Stat1 and the transcriptional activity of NF?B, while JNK regulated the Jak1/Stat1 pathway, and ERK1/2 controlled the expression of Egr-1 and influenced MyD88 dependent MMP-9 expression
Lim et al., Oncogene 1998 : We also show that p38 and JNK1 activated by their upstream kinases induce egr-1 promoter activity through activation of the ternary complex factor, Elk-1