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UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

◀ Back to FGF4

FGF4 — FGF8

Pathways - manually collected, often from reviews:

Text-mined interactions from Literome

Power et al., Dev Dyn 1999 (Abnormalities, Multiple...) : Molecular analysis of RA-deficient limb buds revealed enhanced gli-3 and reduced hoxd-12, hoxd-13, shh, and fgf-4 , while fgf-8 , en-1, and wnt-7a expression remained unaltered
Edom-Vovard et al., Dev Biol 2002 : These results indicate that Fgf4 expressed in muscle is required for the maintenance of scleraxis and tenascin but not Fgf8 expression in tendons
Liu et al., Development 2002 (Limb Deformities, Congenital) : In this system, FGF10 but not FGF8 protein injected into the mutant distal tip mesenchyme restores Fgf8 expression in the AER
Saitsu et al., Mech Dev 2006 (Heart Defects, Congenital) : Moreover, over-expression of Fgf15 resulted in up-regulation of Fgf8 expression in the isthmus/r1
Katayama et al., Int J Oncol 2010 (Prostatic Neoplasms) : Overall, bicalutamide inhibits the cyclin A expression possibly by inhibiting FGF-8 mRNA expression and FGF-8 protein secretion but not by inhibiting FGF receptor ( FGFR ) signalling in androgen dependent cell lines, and by other mechanisms in androgen independent cell lines
Blagovic et al., PloS one 2011 : Then we induce FGF4 signaling in minimal chemically defined medium ( N2B27 ) and inhibit FGF signaling in fully supplemented differentiation medium with cell secreted factors to determine that the non-FGF cell secreted factors are required to promote growth of differentiating mESCs
Haramis et al., Development 1995 : Furthermore, no Fgf-4 transcripts are detected in the ld mutant AER, whereas Fgf-8 transcripts remain expressed
Kettunen et al., Dev Dyn 1998 : This data suggest roles for FGF-8 and FGF-9 during initiation of tooth development, and for FGF-4 and FGF-9 during regulation of tooth shape