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IFNB1 — SOAT1

Text-mined interactions from Literome

Novick et al., J Interferon Cytokine Res 2000 : The 117.7 mAb, representative of this class of receptor nonblocking mAb, induced hyper-tyrosine phosphorylation of IFNAR-2 in the presence of IFN-alpha/beta but did not inhibit IFN-alpha/beta induced Jak-Stat tyrosine phosphorylation and ISGF3 complex formation
Andrejeva et al., J Virol 2002 : In contrast, STAT2 was absent from 2f/PIV2-V cells, and IFN-alpha/beta but not IFN-gamma signaling was blocked in these cells
Parisien et al., J Virol 2002 : Results indicate that neither the IFN-alpha/beta receptor, the tyrosine kinases Jak1 or Tyk2, nor the ISGF3 DNA binding subunit, IFN regulatory factor 9 (IRF9), is required for STAT protein degradation induced by either virus
Nguyen et al., Science 2002 (Arenaviridae Infections) : Here we show that IFN-alpha/beta activate STAT4 directly ( STAT , signal transducers and activators of transcription ) and that this is required for IFN-gamma production during viral infections of mice, in concert with T cell receptor derived signals
Hu et al., J Leukoc Biol 2008 : In this study, we analyzed the regulation of STAT signaling during maturation of human DCs by TNF-alpha and PGE2, which induced maturation of human DCs comparably with LPS but did not induce detectable IFN-beta production or Stat1 tyrosine phosphorylation
Schaber et al., Cancer Res 1998 (Breast Neoplasms) : By analyzing the effect of PRL on IFN alpha/beta induced tyrosine phosphorylation of Stat proteins and their binding to IFN regulated genes, we now report that costimulation of PRL receptors did not interfere with IFN alpha/beta signals in several human breast cancer cell lines, including T47D, MCF-7, and BT-20