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FOS — IL24
Text-mined interactions from Literome
Seppänen et al., Oncol Res 1998
(Adenocarcinoma...) :
In the present study, we have investigated the
effects of interferons-alpha (IFN-alpha) and -gamma ( IFN-gamma ),
interleukin-10 (IL-10) and -13 ( IL-13 ), transforming growth factor-beta1 ( TGF-beta1 ), granulocyte-macrophage colony stimulating factor ( GM-CSF ), and tumor necrosis factor-alpha (TNF-alpha) on cell proliferation and induction of transcription factors
AP-1 and NF-kappaB in UM-EC-3 human endometrial adenocarcinoma cells and UT-OC-5 ovarian carcinoma cells in vitro
Madireddi et al., J Cell Physiol 2000
(Melanoma) :
Employing a heterologous mda-7 luciferase gene reporter system, we demonstrate that ectopic expression of either
AP-1/cJun or C/EBP can significantly
enhance expression of the
mda-7 promoter in melanoma cells
Liacini et al., Matrix Biol 2002
(Osteoarthritis, Hip) :
Inhibition of
interleukin-1 stimulated MAP kinases,
activating protein-1 (AP-1) and nuclear factor kappa B (NF-kappa B) transcription factors down-regulates matrix metalloproteinase gene expression in articular chondrocytes
Wang et al., Nat Immunol 2006
:
Tumor necrosis factor receptor associated factor 6 ( TRAF6 ) is critical for mediating Toll-like receptor ( TLR )
-interleukin 1 receptor (IL-1R) signaling and subsequent
activation of NF-kappaB and
AP-1 , transcriptional activators of innate immunity
Huo et al., Oncol Rep 2013
:
The results showed that
MDA-7/IL-24 overexpression decreased the expression of CD44, ICAM-1, MMP-2/-9, CyclinB, Twist, survivin, TGF-ß and p-Akt, transcriptional activity of NF-?B, and
increased the expression of E-cadherin and p-ERK and transcriptional activity of
AP-1 in HepG2 and BEL-7402 cells
Sung et al., J Biol Chem 1993
:
Stimulation of
interleukin-1 gene transcription may be
caused by the stimulation of transcription factor activities, including those of
AP-1 , by these protein phosphatase inhibitors
Swiergiel et al., Brain Res Bull 1996
:
The role of cerebral noradrenergic systems in the
Fos response to
interleukin-1
Chang et al., Brain Res 1996
:
FOS expression
induced by
interleukin-1 or acute morphine treatment in the rat hypothalamus is attenuated by chronic exposure to morphine