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IL3 — IL8
Text-mined interactions from Literome
Lottaz et al., J Invest Dermatol 2001
:
We hypothesize that inhibition of this pathway prevents secretion of interleukin-1, thereby downregulating
interleukin-1 dependent autocrine induction of
interleukin-8
Zirngibl et al., Mol Cell Biol 2002
:
Bone marrow derived Fps/Fes-null macrophages showed no defects in granulocyte-macrophage colony stimulating factor-,
interleukin 6 (IL-6)-, or
IL-3 induced
activation of signal transducer and activator of transcription 3 ( Stat3 ) and Stat5A or LPS induced degradation of I kappa B or activation of p38, Jnk, Erk, or Akt
Prause et al., Eur J Pharmacol 2003
:
Pharmacological modulation of
interleukin-17 induced GCP-2-, GRO-alpha- and
interleukin-8 release in human bronchial epithelial cells ... We found that
interleukin-17 ( 1-1000 ng/ml )
increased the release of GCP-2, GRO-alpha and
interleukin-8 in a concentration dependent manner
Waehre et al., J Am Coll Cardiol 2003
(Coronary Artery Disease) :
Our main findings were : 1 ) gene expression of several chemokines ( i.e., macrophage inflammatory protein [MIP ] -1alpha, MIP-1beta, and
interleukin [ IL]-8 ) and chemokine receptors ( i.e., CC chemokine receptor [ CCR]1, CCR2, CCR4, and CCR5 ) was markedly increased among CAD patients compared with healthy control subjects ; 2 ) treatment with atorvastatin and simvastatin markedly reduced the mRNA levels of some of these chemokines ( i.e., MIP-1alpha, MIP-1beta, IL-8 ) and receptors ( i.e., CCR1 and CCR2 ), with the most pronounced effect in the atorvastatin group ; and 3 ) statin therapy
reduced the spontaneous release of
IL-8 and MIP-1alpha from PBMCs in CAD patients
Vittori et al., J Allergy Clin Immunol 1992
:
Protective effect of nedocromil sodium on the
interleukin-1 induced production of
interleukin-8 in human bronchial epithelial cells
Uchiyama et al., Am J Obstet Gynecol 1992
:
These results indicate that
interleukin-8-like chemotactic factor participates in the cervical ripening at term pregnancy and that the production of this factor is
controlled effectively by
interleukin-1
Soloff et al., Endocrinology 2004
:
In situ analysis of
interleukin-1 induced transcription of cox-2 and
il-8 in cultured human myometrial cells
Wuyts et al., J Heart Lung Transplant 2004
:
N-acetylcysteine inhibits
interleukin-17 induced
interleukin-8 production from human airway smooth muscle cells : a possible role for anti-oxidative treatment in chronic lung rejection ?
Hirota et al., Rheumatol Int 1992
:
Production of
interleukin 8 by cultured synovial cells in
response to
interleukin 1 and tumor necrosis factor
Matsuzaki et al., Cancer Sci 2006
:
We have demonstrated previously that the Th1-cytokine conditioned bone marrow derived dendritic cell ( BMDC ) subset BMDC1 ( generated in the presence of granulocyte-macrophage colony stimulating factor [GM-CSF ]
+ interleukin [ IL]-3 + interferon [ IFN]-gamma+ IL-12 )
induces a much stronger type 1 immune response than BMDC0 ( GM-CSF +
IL-3 )
Hashmi et al., Coron Artery Dis 2006
(Angina, Unstable) :
Role of interleukin-17 and
interleukin-17 induced cytokines interleukin-6 and
interleukin-8 in unstable coronary artery disease
Cohen et al., Immunol Lett 1991
:
We have studied the production of interleukin-1 (IL-1),
interleukin-6 (IL-6) and tumor necrosis factor (TNF) by mouse peritoneal macrophages triggered by lipopolysaccharide (LPS) in the
presence or absence of
IL-3
Darbonne et al., J Clin Invest 1991
:
Their binding of IL-8 is rapidly reversible and does not result in receptor internalization, although bound IL-8 is resistant to extraction by pH 3 buffer at 5 degrees C.
125I-IL-8 binding to red cells was not
inhibited by epidermal growth factor or
interleukin 1, but was inhibited by monocyte chemotactic peptide-1, which is not a neutrophil chemotaxin, but is a member of the same family of polypeptides as IL-8
Larsen et al., Biochem Biophys Res Commun 1991
:
1,25 ( OH ) 2-D3 is a potent regulator of
interleukin-1 induced
interleukin-8 expression and production
Hua et al., Molecular oral microbiology 2010
(Periodontal Diseases) :
Furthermore, as little as 2 µg ml(-1) mPE was sufficient to inhibit
interleukin-1ß induced secretion of
interleukin-8 in both gingival epithelial cells and THP-1 cells
Vindeløv et al., Growth Horm IGF Res 2011
(Adenoma...) :
Interleukin-8 production from human somatotroph adenoma cells is
stimulated by
interleukin-1ß and inhibited by growth hormone releasing hormone and somatostatin
Strieter et al., Am J Respir Cell Mol Biol 1990
:
The
induction of AMO derived
IL-8 by tumor necrosis factor (TNF), lipopolysaccharide (LPS), and
interleukin-1 ( IL-1 beta ) was shown to be both dose and time dependent
Cavaillon et al., Cell Immunol 1990
:
Induction of
interleukin-3 by
interleukin-1 in the absence of other exogenous stimuli
Korpelainen et al., Proc Natl Acad Sci U S A 1993
(Inflammation) :
IL-3 binding to TNF-alpha activated HUVEC
enhanced IL-8 production, E-selection expression, and neutrophil transmigration
Arnold et al., Immunology 1994
(Respiratory Syncytial Virus Infections) :
In addition,
IL-3 , G-CSF, IFN-gamma and TGF-beta 2, albeit at high concentrations,
induced a low
IL-8 release from non infected A549 cells
Takahashi et al., Blood 1993
:
Effect of granulocyte-macrophage colony stimulating factor and
interleukin-3 on
interleukin-8 production by human neutrophils and monocytes
Kita et al., C R Seances Soc Biol Fil 1994
(Stomach Neoplasms) :
Although IL-1 beta, IL-4, IL-5, IL-6, IL-7,
IL-8 , TNF-alpha, IFN-alpha and IFN-gamma mRNA were detected in all the samples tested,
IL-3 and IFN-beta mRNA was not
detected at all
Ng et al., J Biol Chem 1994
:
Although tumor necrosis factor (TNF) and
interleukin-1 ( IL ) induce the expression of the
IL-8 , TNF stimulated gene 6, and plasminogen activation inhibitor-2 genes and the activation of NF-kappa B with nearly identical kinetics, the two cytokines differ significantly in the induction of the IL-6 gene in all primary fibroblasts tested
Van Hoffen et al., Am J Pathol 1996
:
Using immunohistochemistry, IL-2,
IL-3 , and IL-10 protein was
detected in biopsies with high rejection grades, whereas few cells expressed IL-6,
IL-8 , and IFN-gamma
Miyamasu et al., Allergy 1997
:
IL-3 also
potentiated C5a induced
IL-8 generation
Rougier et al., Cytokine 1998
:
IL-1 alpha, IL-1 beta, and
IL-3 , tumour necrosis factor alpha (TNF-alpha), Stem cell factor (SCF) and granulocyte-macrophage colony stimulating factor ( GM-CSF ) ( at 10 ng/ml )
stimulated IL-6 and
IL-8 production in 0 % and 10 % FCS
Krakauer et al., J Infect Dis 1998
:
Interleukin-8 production by human monocytic cells in response to staphylococcal exotoxins is direct and
independent of
interleukin-1 and tumor necrosis factor-alpha