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ITGAL — TNF

Text-mined interactions from Literome

Hideshima et al., Oncogene 2001 (MAP Kinase Signaling System...) : TNFalpha in both a dose and time dependent fashion induced expression of CD11a ( LFA-1 ), CD54 ( intercellular adhesion molecule-1, ICAM-1 ), CD106 ( vascular cell adhesion molecule-1, VCAM-1 ), CD49d ( very late activating antigen-4, VLA-4 ), and/or MUC-1 on MM cell lines ; as well as CD106 ( VCAM-1 ) and CD54 ( ICAM-1 ) expression on BMSCs
Rezaie-Majd et al., Arterioscler Thromb Vasc Biol 2003 (Hypercholesterolemia) : Moreover, simvastatin, atorvastatin, and cerivastatin were found to downregulate tumor necrosis factor (TNF)-alpha induced expression of CD54 and CD18/CD11a in isolated PBMCs obtained from normal donors as well as TNF-alpha dependent expression of these CAMs in cultured human umbilical vein endothelial cells ( HUVECs )
Yamaguchi et al., Infect Immun 2004 : Northern and Western blotting analyses showed that TNF-alpha enhanced the expression of CD11a in the cells at both the mRNA and protein levels but did not do so for CD18 expression
Tang et al., Am J Physiol 1995 : In conclusion, 1 ) CD11a , ICAM-1, and CINC play major roles in the LPS initiated emigration of PMNs into the bronchoalveolar space, and 2 ) the TNF that drives ICAM-1 and CINC expression is derived largely from alveolar macrophages rather than PMNs
Aramburu et al., J Immunol 1993 : It is noteworthy that the Kp43 mediated production of TNF-alpha was partially inhibited by anti-CD18, anti-CD11a and anti-ICAM-1 mAb that blocked LFA-1 dependent cellular interactions, which impaired NK cell aggregation and, moreover, was dependent on the presence of extracellular Mg2+, thus suggesting that the leukocyte integrin is involved in the activation process triggered through Kp43
Ferrante et al., J Immunol 1993 : TNF increased the expression of CR3 ( CD11b/CD18 ) and CR4 ( P150, 95 ; CD11c/CD18 ) adhesion receptors but not LFA-1 ( CD11a/CD18 ) ; and mAb against the alpha-chain of either CR3 or CR4 but not LFA-1 prevented the enhancing effects of TNF on the neutrophil bactericidal activity
Takeshita et al., Infect Immun 1998 : Furthermore, Northern blot analyses showed that the fimbria induced expression of IL-1beta and TNF-alpha genes in the cells was inhibited strongly by CD18 antibody treatment and slightly by CD11a , CD11b, or CD11c antibody treatment