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CALM3 — PRKACB

Text-mined interactions from Literome

Galisteo et al., J Pharmacol Exp Ther 1999 : Taken together, our results suggest that CYP3A induction by PB is regulated positively by calmodulin dependent protein kinase and cGMP dependent protein kinase, and negatively by PKA in mouse hepatocytes in primary culture
Tzavara et al., Eur J Neurosci 2000 (Substance Withdrawal Syndrome) : We show that after SR141716A precipitation of cannabinoid withdrawal, basal and calcium-calmodulin stimulated adenylyl cyclase activities as well as active PKA in the cerebellum increase in a transient manner with a temporal profile which matches that of the somatic expression of abstinence
Tang et al., Acta Pharmacol Sin 2000 : To study the effects of dopamine ( DA ) and glutamate ( Glu ) and their receptor agonists/antagonists on Ca2+/calmodulin dependent protein kinase II ( CCDPK II ), cyclic AMP dependent protein kinase A (PKA) activities and the LDH release in rat striatal slices, and to examine the interaction between DA and Glu transmitter systems in striatum
Kudoh et al., Brain Res 2001 (Magnesium Deficiency...) : In this paper, protein kinase C ( PKC ) and calcium/calmodulin dependent protein kinase type II ( CaMKII ) but not protein kinase A (PKA) were reported to contribute to the potentiation mechanism during a step between the activation of the N-methyl-D-aspartate receptors by the Mg ( 2+ ) -free treatment and the secretion of the protein factor ( s )
Nesher et al., Diabetes 2002 (Diabetes Mellitus, Type 2) : We suggest that three families of PKs ( calmodulin dependent PK [ CaMK ], PKA , and PKC ) function as distal amplifiers for stimulus-secretion coupling signals originating from fuel metabolism, as well as from incretins acting through membrane receptors, adenylate cyclase, and phospholipase C
Kawaguchi et al., J Neurosci 2002 : We confirmed that inhibition of Ca ( 2+ ) /calmodulin dependent protein kinase II ( CaMKII ) or PKA suppresses RP
Sorensen et al., Mol Pharmacol 2002 (Second Messenger Systems) : In the present study, we report that in addition to protein kinase C ( PKC ), cAMP dependent protein kinase (PKA) and cGMP dependent protein kinase ( PKG ) can inhibit calmodulin (CaM) interactions with the carboxyl-terminal tail of mGluR7
Kotak et al., J Neurobiol 2002 : In this study, we asked whether the calcium dependency of inhibitory depression was associated with the activation of calcium/calmodulin dependent protein kinase II ( CaMKII ), protein kinase C ( PKC ), and/or cAMP dependent protein kinase A (PKA)
Yang et al., J Neurophysiol 2004 : Considerable evidence has shown that calcium/calmodulin dependent protein kinase II ( CaMKII ), protein kinase A (PKA) , and protein kinase C ( PKC ) are important for LTP in hippocampus
Ciarimboli et al., J Cell Physiol 2004 : hOCT1 expressed in CHO-cells was inhibited by protein kinase A (PKA) activation ( 1 microM forskolin, -58 +/- 6 %, n = 12 ), calmodulin inhibition ( 0.1 microM calmidazolium, -68 +/- 3 %, n = 6 ; 10 microM ophiobolin A, -48 +/- 10 %, n = 7 ), calmodulin dependent kinase II inhibition ( 1 microM KN62, -78 +/- 4 %, n = 12 ), and inhibition of p56 ( lck ) tyrosine kinase ( 10 microM aminogenistein, -35 +/- 7 %, n = 12 )
Rakhilin et al., Science 2004 (Calcium Signaling) : G protein coupled receptor ( GPCR ) -dependent activation of protein kinase A (PKA) led to phosphorylation of RCS at Ser55 and increased its binding to CaM
Hongpaisan et al., J Neurosci 2004 (Calcium Signaling) : Large increases in cytosolic free Ca2+ ( [ Ca2+ ] i ) activate several kinases that are important for neuronal plasticity, including Ca2+/calmodulin dependent kinase II ( CaMKII ), protein kinase A (PKA) , and protein kinase C ( PKC )
Zheng et al., Pharmacol Ther 2005 (Heart Failure) : More importantly, persistent beta1AR stimulation evokes a multitude of cardiac toxic effects, including myocyte apoptosis and hypertrophy, via a calmodulin dependent protein kinase II ( CaMKII ) -, rather than cAMP-PKA- , dependent mechanism in rodent heart in vivo and cultured cardiomyocytes
Huang et al., J Neurosci 2006 : These Ng-/- mice, as compared with Ng+/+, respond poorly after treatment of their hippocampal slices with agents that activate signaling molecules important for learning and memory, including Ca2+/calmodulin dependent protein kinase II ( alphaCaMKII ), PKC, protein kinase A (PKA) , extracellular signal regulated kinase ( ERK ), and cAMP response element binding protein ( CREB )
Siarey et al., J Neurochem 2006 (Down Syndrome) : Here we report that calcium/calmodulin dependent protein kinase II ( CaMKII ), phosphatidylinositol 3-kinase (PI3K)/Akt, extracellular signal regulated kinase ( ERK ), protein kinase A (PKA) and protein kinase C ( PKC ), all of which have been shown to be involved in synaptic plasticity, are altered in the Ts65Dn hippocampus
D'Angiolella et al., PloS one 2007 : MPF down-regulation required Ca ( ++ ) /calmodulin dependent kinase II ( CaMKII ) and cAMP dependent protein kinase (PKA) activities at meiosis and mitosis exit, respectively
Dash et al., Learn Mem 2007 (Memory Disorders) : Additionally, the negative influence of calcium- and cAMP dependent protein kinase ( i.e., calcium/calmodulin dependent protein kinase II ( CaMKII ), calcium/diacylglycerol activated protein kinase C ( PKC ), and cAMP dependent protein kinase A (PKA) ) activities on working memory will be reviewed
Machado et al., J Neurosci Res 2008 (MAP Kinase Signaling System) : Inhibiting calcium/calmodulin dependent kinase II ( CaMKII ), calcineurin, or protein kinase A (PKA) activity prevents KCl induced IPD-IEG mRNA accumulation, suggesting that the IPD-IEG genes are induced by depolarization in neurons via a combination of calcineurin/PKA- and CaMKII dependent pathways
Boudreau et al., J Neurochem 2009 (Cocaine-Related Disorders...) : We focused on calcium/calmodulin dependent protein kinase II ( CaMKII ), extracellular signal regulated protein kinase ( ERK ), and protein kinase A (PKA)
Ogrodnik et al., J Physiol 2010 (Calcium Signaling) : This was likely to have been mediated by Ca ( 2+ ) /calmodulin dependent protein kinase ( CaMKII ), rather than cAMP dependent protein kinase (PKA)
Song et al., J Biol Chem 2010 : We found that Ser-21/9 phosphorylation of GSK-3 was mediated by Ca ( 2+ ) /calmodulin dependent protein kinase II ( CaMKII ) but not by Akt/PKB, PKA , or p90 ( RSK )
Soltis et al., Biophys J 2010 (Arrhythmias, Cardiac...) : Cardiac excitation-contraction coupling is a highly coordinated process that is controlled by protein kinase signaling pathways, including Ca ( 2+ ) /calmodulin dependent protein kinase II ( CaMKII ) and protein kinase A (PKA) ... To better understand mechanisms of cardiac CaMKII function, we integrated dynamic CaMKII dependent regulation of key Ca ( 2+ ) handling targets with previously validated models of cardiac EC coupling, Ca ( 2+ ) /calmodulin dependent activation of CaMKII, and ß-adrenergic activation of PKA
McArdle et al., J Virol 2011 : We find that inhibition of calmodulin dependent kinase kinase ( CaMKK ), but not calmodulin dependent kinase II ( CaMKII ) or protein kinase A (PKA) , blocks HCMV mediated activation of glycolysis
Moncada et al., Proc Natl Acad Sci U S A 2011 : Moreover, activation of the glutamatergic NMDA receptors is required for setting the learning tags, and this machinery further required a-Ca ( 2+ ) /calmodulin dependent protein kinase II and PKA but not ERK1/2 activity
Chiluiza et al., J Biol Chem 2013 (Glomerulosclerosis, Focal Segmental) : However, these effects appear to be independent of their effects on calcium/calmodulin dependent protein kinase II and PKA , respectively
Tan et al., J Neurosci 1994 : In this study 32P labeling of non-NMDA GluRs was investigated in cultured hippocampal neurons stimulated 2-15 min with agonists that selectively stimulate either Ca2+/calmodulin dependent protein kinase II ( CaM-kinase II ), Ca2+/phospholipid dependent protein kinase C ( PKC ), or cAMP dependent protein kinase A (PKA)
Menez et al., Alcohol Alcohol 1993 : Phosphorylation of pure cytochrome P4502E1 (CYP2E1) was achieved in vitro using Ca2+/calmodulin dependent protein kinase II ( CaM kinase II ), protein kinase C ( PKC ) and cAMP dependent protein kinase (PKA)