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MET — PTK6
Text-mined interactions from Literome
Suzuki et al., Biochim Biophys Acta 2002
(Chondrosarcoma) :
High molecular weight HA is inactive, but lower molecular weight fragments ( M ( r ) 3.5 kDa ) are active with maximal effect in the microg/ml range ; ( d ) the standard form of CD44 ( CD44s ) is critical for the response because the effect on c-Met, both in terms of upregulation and phosphorylation, is inhibited by preincubation with an anti-CD44 monoclonal antibody ; and ( e ) phosphorylation of
c-Met induced by CD44 stimulation is
inhibited by
protein tyrosine kinase inhibitor, tyrphostin
Castro et al., Breast Cancer Res 2010
(Breast Neoplasms) :
Breast tumor kinase and extracellular signal regulated kinase 5
mediate Met receptor signaling to cell migration in breast cancer cells