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JUN — RAN

Text-mined interactions from Literome

Qiao et al., J Biol Chem 2010 (Hypersensitivity, Delayed) : One of the possible mechanisms is that intracellular Ran protein levels control the nuclear retention for selective transcription factors such as c-Jun and c-Fos of AP-1 , which is known to be critical in T cell activation and proliferation and lymphokine secretion
Milano et al., J Biol Chem 2012 (Cell Transformation, Neoplastic) : Furthermore, Ran ( K152A ) expression in the human mammary SKBR3 adenocarcinoma cell line gives rise to an enhanced transformed phenotype and causes a robust stimulation of both ERK and the N-terminal c-Jun kinase (JNK)