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PRKAR2B — TCF19

Text-mined interactions from Literome

Hino et al., Mol Cell Biol 2005 : Ser675 was found to be a site for phosphorylation by PKA, and substitution of this serine residue with alanine in beta-catenin attenuated inhibition of the ubiquitination of beta-catenin by PKA, PKA induced stabilization of beta-catenin, and PKA dependent activation of Tcf
Ponsioen et al., Exp Cell Res 2007 : This degree of cAMP transfer sufficed to evoke a well characterized response to cAMP in neighbor cells, i.e. the PKA mediated phosphorylation of the ER transcription factor in A431 carcinoma cells
O'Driscoll et al., J Neurochem 2007 : Activation of the transcription factor cAMP-response element binding protein ( CREB ) and subsequent up-regulation of Bcl-2 in response to bFGF was also dependent on PKA as inhibition with H-89 attenuated increased pCREB levels and Bcl-2 expression
Taurin et al., Am J Physiol Cell Physiol 2008 : This translates to a PKA dependent stimulation of TCF transcriptional activity through an increased association of phosphorylated ( by PKA ) beta-catenin with TCF-4
Ren et al., Genomics 2009 : Mutagenesis and cotransfection experiments showed that PKA regulation of this c-fos intronic element was mediated by two adjacent CRE-like sequences and the transcription factor CREB
Oeseburg et al., Arterioscler Thromb Vasc Biol 2010 (Diabetes Mellitus...) : Further analysis revealed that GLP-1 activates the cAMP response element binding ( CREB ) transcription factor in a cAMP/protein kinase A (PKA) dependent manner, and inhibition of the cAMP/PKA pathway abolished the GLP-1 protective effect
Yen et al., J Biol Chem 2011 (MAP Kinase Signaling System) : PKA , PI3K, and ERK inhibitors abolished PGE2- and cAMP induced c-Fos and MMP-9 up-regulation, and ERK activation was required for the binding of activator protein 1 (AP-1) transcription factor to the MMP-9 promoter
Andersen et al., Development 2012 : In addition, we show that centrosome position and motility are regulated by LIM homeodomain transcription factor activity, which is specifically required for the development of RB peripheral axons
Su et al., PloS one 2012 : In the present studies, we have demonstrated that PKA enhances NF-E2 SUMOylation in an in vitro system using purified proteins, suggesting a possible mechanism for PKA dependent activation of the NF-E2 transcription factor through SUMOylation
Hagiwara et al., Mol Cell Biol 1993 : Cyclic AMP ( cAMP ) regulates a number of eukaryotic genes by mediating the protein kinase A (PKA) dependent phosphorylation of the CREB transcription factor at Ser-133
Richards et al., J Biol Chem 1996 : The transcription factor CREB ( cAMP responsive element binding protein ) is activated by protein kinase A (PKA) phosphorylation of a single serine residue
Jaspers et al., J Cell Physiol 1998 (Adenocarcinoma, Bronchiolo-Alveolar...) : Exposure to ozone increased interleukin 8 expression and transcription factor activities in a protein tyrosine kinase ( PTK ) -dependent and protein kinase A (PKA) dependent , yet protein kinase C ( PKC ) -independent, manner
Zanger et al., Mol Endocrinol 1999 : The pituitary-specific transcription factor , Pit-1, is necessary to mediate protein kinase A (PKA) regulation of the GH, PRL, and TSH-beta subunit genes in the pituitary