Boon et al., Cancer Sci 2006
(Intestinal Neoplasms) :
Activation of Wnt signaling in the intestinal mucosa of Apc +/min mice does not cause overexpression of the receptor tyrosine kinase Met Boyle et al., Development 2011
:
Only nephron stem cells have the ability to undergo MET in response to Wnt or Notch, as activation in the closely related stromal mesenchyme has no inductive effect
Kim et al., Neuro Oncol 2013
(Brain Neoplasms...) :
Conclusion These data suggest that Wnt/ß-catenin signaling is a key downstream effector of MET signaling and contributes to the maintenance of GSC and GBM malignancy