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APOB — CSF1
Text-mined interactions from Literome
Fuhrman et al., Atherosclerosis 2008
:
We thus conclude that
Ox-LDL induces monocyte differentiation to macrophages in vivo and this phenomenon
involves activation of the
M-CSF-receptor
Ishibashi et al., J Biol Chem 1990
:
Binding of
LDL was also
enhanced by
M-CSF but less significantly than that of acetyl-LDL
Rajavashisth et al., Arterioscler Thromb Vasc Biol 1995
:
Nuclear runon assays showed that
M-CSF gene transcription was
activated by
MM-LDL
Watanabe et al., Ann N Y Acad Sci 1995
(Arteriosclerosis) :
M-CSF ( 100 ng/ml ) enhanced acidic and neutral CE hydrolase, and ACAT activities by 3.2-fold, 4-fold, and 2.3-fold, respectively, in the
presence of acetyl
LDL , and
M-CSF increased ratios of both acidic and neutral CE hydrolase activities to ACAT activity
Suzu et al., J Clin Invest 1994
(Arteriosclerosis) :
The addition of exogenous chondroitin sulfate or apolipoprotein B inhibited the binding of PG-M-CSF to LDL in a dose dependent manner, indicating that the interaction between
PG-M-CSF and LDL was
mediated by the binding of the chondroitin sulfate chain of PG-M-CSF to LDL
apolipoprotein B
Pai et al., Kidney Int 1995
:
Since atherogenic lipoproteins ( such as low density lipoprotein, LDL ) have been implicated in monocyte migration and proliferation into the glomerular mesangium, we examined the
effect of
LDL on mesangial cell expression of macrophage colony stimulating factor (
M-CSF ), a cytoregulatory peptide associated with monocyte chemoattraction, differentiation and proliferation