Sancho et al., Mol Pharmacol 2003
:
The NF-kappaB inhibitory activity of AEA was independent of CB(1) and CB(2) activation in TNFalpha stimulated 5.1 and A549 cell lines, which do not express vanilloid receptor 1, and was not mediated by hydrolytic products formed through the activity of the enzyme fatty acid amide hydrolase
Bátkai et al., Am J Physiol Heart Circ Physiol 2007
(Inflammation...) :
Anandamide dose dependently attenuated the TNF-alpha induced ICAM-1 and VCAM-1 expression, NF-kappaB activation in HCAECs, and the adhesion of monocytes to HCAECs in a CB(1)- and CB(2) dependent manner