Gene interactions and pathways from curated databases and text-mining

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CSF3 — IL8

Text-mined interactions from Literome

Akahane et al., Arzneimittelforschung 1990 : The enhanced G-CSF production from NFSA cells may be due at least to interleukin-1 released from muroctasin stimulated macrophages
Leizer et al., Blood 1990 : Cytokine regulation of colony stimulating factor production in cultured human synovial fibroblasts : I. Induction of GM-CSF and G-CSF production by interleukin-1 and tumor necrosis factor
Jiang et al., J Int Med Res 2010 (Alzheimer Disease...) : Levels of a7 nAChR protein were significantly increased and levels of interleukin-1ß , tumour necrosis factor-a and nuclear factor-?B ( NF-?B ) protein were significantly decreased in the brain of APP transgenic mice in response to G-CSF
Wang et al., Int J Oncol 1995 : We report that all-trans retinoic acid ( tRA ) synergizes with LPS to enhance the production of granulocyte colony stimulating factor ( G-CSF ) in PMA treated cells, whereas the production of granulocyte-macrophage CSF, interleukin 1-beta (IL-1-beta), and tumor necrosis factor-alpha (TNF-alpha) is minimally affected by tRA
Lloyd et al., J Biol Chem 1995 : Similarly, 125I-IL-8 ligand binding to PMN is increased by G-CSF and decreased by LPS treatment ... The stimulatory effect of G-CSF on IL-8R expression is transcriptional as it is inhibited by actinomycin D and is evident in nuclear run-on analyses
Arnold et al., Immunology 1994 (Respiratory Syncytial Virus Infections) : In addition, IL-3, G-CSF , IFN-gamma and TGF-beta 2, albeit at high concentrations, induced a low IL-8 release from non infected A549 cells
Pajkrt et al., Blood 1997 (Endotoxemia) : In group 1, G-CSF significantly enhanced the release of tumor necrosis factor (TNF), interleukin-6 (IL-6), IL-8, IL-1 receptor antagonist (IL-1ra), and soluble TNF receptors
Rougier et al., Cytokine 1998 : G-CSF stimulated and IL-4 inhibited IL-8 production in 10 % FCS