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FOS — MYLIP
Text-mined interactions from Literome
Talotta et al., Oncogene 2009
(Cell Transformation, Neoplastic) :
Here, we show that the miRNA
miR-21 , which represents the most frequently upregulated oncomir in solid tumors, is
induced by
AP-1 in response to RAS
Tili et al., Carcinogenesis 2010
:
We further establish that the downregulation of AP-1 activity by resveratrol is
miR-663 dependent and that the effects of resveratrol on both
AP-1 activity and JunB levels are dose
dependent
Dai et al., Hum Reprod 2011
(Pre-Eclampsia) :
DNA precipitation assays and luciferase reporter analysis were used to evaluate the
regulation of
miR-155 by
AP-1 and NF-?B
Zhou et al., Proc Natl Acad Sci U S A 2011
(Inflammation) :
Overexpression of PPARa significantly attenuated the
AP-1 mediated
miR-21 expression ... These results demonstrate a unique mechanism by which OSS induces
AP-1 dependent
miR-21 expression, which directly targets PPARa to inhibit its expression, thereby allowing activation of AP-1 and the promotion of monocyte adhesion
Wu et al., J Lipid Res 2012
(Hyperlipidemias) :
Thus, abundance of miR-181a reduced c-Fos protein, whereas inhibition of
miR-181a increased
c-Fos protein in BMDCs
Fan et al., Cell Biochem Funct 2013
(Carcinoma, Hepatocellular...) :
miR-139 suppressed the luciferase activity of the
pGL-FOS by approximately 40 % compared with the negative control
Song et al., PloS one 2012
:
Stretch upregulates miR-21 expression at least in part at the transcription level and
AP-1 is
essential for stretch induced
miR-21 expression
Rossi et al., J Cell Physiol 2013
(Bone Resorption...) :
Moreover, we found that canonical targets
C-FOS and metalloproteinase 2 are
suppressed by constitutive
miR-29b expression which also downregulated the master OCL transcription factor, NAFTc-1
Onyeagucha et al., Exp Cell Res 2013
(Colonic Neoplasms) :
Altogether, these data demonstrate that the expression of
miR-155 is regulated by S100P and is
dependent on RAGE activation and stimulation of
AP-1
Kang et al., Mol Biol Rep 2013
:
miR-21 could significantly
promote adipocyte differentiation, increase adiponectin mRNA and protein expression, while decrease
AP-1 protein level
Tao et al., Eur J Cancer 2013
(Disease Progression...) :
Involvement of
FOS mediated
miR-181b/miR-21 signalling in the progression of malignant gliomas ... Reduction of
FOS inhibited
miR-21 expression by binding to the miR-21 promoter using luciferase reporter assays