Gene interactions and pathways from curated databases and text-mining

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FOS — MYLIP

Text-mined interactions from Literome

Talotta et al., Oncogene 2009 (Cell Transformation, Neoplastic) : Here, we show that the miRNA miR-21 , which represents the most frequently upregulated oncomir in solid tumors, is induced by AP-1 in response to RAS
Tili et al., Carcinogenesis 2010 : We further establish that the downregulation of AP-1 activity by resveratrol is miR-663 dependent and that the effects of resveratrol on both AP-1 activity and JunB levels are dose dependent
Dai et al., Hum Reprod 2011 (Pre-Eclampsia) : DNA precipitation assays and luciferase reporter analysis were used to evaluate the regulation of miR-155 by AP-1 and NF-?B
Zhou et al., Proc Natl Acad Sci U S A 2011 (Inflammation) : Overexpression of PPARa significantly attenuated the AP-1 mediated miR-21 expression ... These results demonstrate a unique mechanism by which OSS induces AP-1 dependent miR-21 expression, which directly targets PPARa to inhibit its expression, thereby allowing activation of AP-1 and the promotion of monocyte adhesion
Wu et al., J Lipid Res 2012 (Hyperlipidemias) : Thus, abundance of miR-181a reduced c-Fos protein, whereas inhibition of miR-181a increased c-Fos protein in BMDCs
Fan et al., Cell Biochem Funct 2013 (Carcinoma, Hepatocellular...) : miR-139 suppressed the luciferase activity of the pGL-FOS by approximately 40 % compared with the negative control
Song et al., PloS one 2012 : Stretch upregulates miR-21 expression at least in part at the transcription level and AP-1 is essential for stretch induced miR-21 expression
Rossi et al., J Cell Physiol 2013 (Bone Resorption...) : Moreover, we found that canonical targets C-FOS and metalloproteinase 2 are suppressed by constitutive miR-29b expression which also downregulated the master OCL transcription factor, NAFTc-1
Onyeagucha et al., Exp Cell Res 2013 (Colonic Neoplasms) : Altogether, these data demonstrate that the expression of miR-155 is regulated by S100P and is dependent on RAGE activation and stimulation of AP-1
Kang et al., Mol Biol Rep 2013 : miR-21 could significantly promote adipocyte differentiation, increase adiponectin mRNA and protein expression, while decrease AP-1 protein level
Tao et al., Eur J Cancer 2013 (Disease Progression...) : Involvement of FOS mediated miR-181b/miR-21 signalling in the progression of malignant gliomas ... Reduction of FOS inhibited miR-21 expression by binding to the miR-21 promoter using luciferase reporter assays