Gene interactions and pathways from curated databases and text-mining

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FOS — PDCD11

Text-mined interactions from Literome

Lehmann et al., Cell Death Differ 2002 : This study demonstrates a role for AP-1 in the stage-specific steroid triggered programmed cell death of larval tissues during Drosophila metamorphosis
Zahradnícková et al., Cell Mol Life Sci 2003 (Cell Transformation, Neoplastic...) : c-Fos but not v-Fos protein induces programmed cell death of v-myb transformed monoblasts ... We show that while the v-Fos protein prolongs the G0G1 phase of the BM2 cell cycle, c-Fos leaves the cell cycle unaffected and, rather, induces programmed cell death
Zhu et al., Int J Biochem Cell Biol 2012 (Bone Resorption) : The RANKL stimulated mRNA expression of osteoclast related genes and transcription factors were also diminished by ISL. Mechanistically, ISL blocked the RANKL triggered RANK-TRAF6 association, phosphorylation of mitogen activated protein kinases ( MAPKs ), inhibitor of ?Ba ( I?Ba ) phosphorylation and degradation, nuclear factor-?B ( NF-?B ) p65 nuclear translocation, as well as activator protein (AP)-1 activation
Marti et al., Oncogene 1994 : A similar induction of AP-1 ( cFos/JunD ) was also observed in the involuting rat ventral prostate pointing to a possible role for AP-1 in programmed cell death
Feng et al., Oncogene 1998 (Atrophy) : Our data strongly suggest that c-Fos is required for programmed cell death of prostate epithelial cells