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GNRH1 — NOS1

Text-mined interactions from Literome

Gobbetti et al., Biol Reprod 1999 : GnRH increased NOS activity during pre-reproduction, noncourtship, and recovery
Bachir et al., Endocrinology 2001 : These data provide evidence for the presence of several elements controlling NOS I expression in gonadotrophs and demonstrate that GnRH , the prime regulator of gonadotrope function, and cAMP may induce the transcription of NOS I in these cells
Tsumori et al., Endocrine 2002 : These findings suggest that GnRH and GHRH could activate nNOS in the gonadotrophs and the somatotrophs, respectively
Wang et al., Fertil Steril 2006 (Endometriosis...) : To determine whether endometrial expression of endothelial nitric oxide synthase (eNOS) or inducible NOS (iNOS) protein in women with endometriosis associated infertility is different from that in the fertile controls, whether GnRH agonist (GnRH-a) regulates the endometrial expression of NOS in women with endometriosis associated infertility, and whether there is a correlation between serum E2 or P levels, and the endometrial expression of eNOS or iNOS
Reynoso et al., Exp Clin Endocrinol Diabetes 2007 : Since glutamate ( GLU ) and GABA are involved in the hypothalamic control of Gn-RH neurons and also in the neuroendocrine mechanism of puberty, in a second serie of experiments, we evaluated the effect of a competitive inhibitor of nitric oxide synthase (NOS) , N-monomethyl-L-arginine ( NMMA ) on Gn-RH , GLU and GABA release in response to leptin
Chang et al., Gen Comp Endocrinol 2010 : In goldfish, nitric oxide synthase (NOS) immunoreactivity is present in gonadotropes and extracellular signal regulated protein kinase ( ERK ) mediates GnRH stimulation of gonadotropin release and synthesis ... In goldfish, nitric oxide synthase (NOS) immunoreactivity is present in gonadotropes and extracellular signal regulated protein kinase ( ERK ) mediates GnRH stimulation of gonadotropin release and synthesis
Garrel et al., Biol Reprod 2010 : Previous in vivo studies have established that pituitary nitric oxide synthase type 1 (NOS1) is regulated by gonadotropin releasing hormone (GnRH) ... In primary cultures of rat pituitary cells, sustained stimulation with the GnRH agonist triptorelin (GnRHa) increased NOS1 protein levels, whereas NOS2 and NOS3 levels were unaffected ... Overall, these experiments demonstrated that GnRH induced up-regulation of pituitary NOS1 is mediated notably by the cAMP/PKA pathway
Garrel et al., Endocrinology 1998 : Expression in gonadotrophs of a GnRH dependent NOS I and the ensuing production of nitric oxide represents a potentially novel signaling pathway for the neuropeptide in the anterior pituitary, consistent with the previously reported GnRH induced cGMP production, the role of which remains to be evaluated
Lozach et al., Mol Cell Endocrinol 1998 : GnRH dependent up-regulation of nitric oxide synthase I level in pituitary gonadotrophs mediates cGMP elevation during rat proestrus ... To further establish the GnRH regulation of pituitary NOS I under physiological circumstances, we have examined the expression of NOS I during the 4-day estrus cycle in rats