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CSF1 — GPI
Text-mined interactions from Literome
Pixley et al., Mol Cell Biol 2001
:
Here we have overexpressed wild-type ( WT ) PTP
phi and a phosphatase-inactive ( C325S ) mutant in a macrophage cell line in the
presence and absence of
CSF-1 ... In the
presence of
CSF-1 , WT PTP
phi induces cell rounding and ruffle formation, while C325S PTP phi has no effect
Lenda et al., J Immunol 2003
(Inflammation...) :
CSF-1 is
required for M
phi proliferation and survival ... We suggest that during renal inflammation,
CSF-1 mediates M
phi recruitment, proliferation, activation, and, in turn, TEC apoptosis
Bouyer et al., Endocrinology 2007
:
Although a rise in intracellular pH ( pH(i) ) has been linked to inhibition of apoptosis, the
effect of
CSF-1 on
pH(i) in osteoclasts has not been reported ... The present study shows that, in the absence of CO ( 2 ) /HCO ( 3 ) ( - ),
CSF-1 causes little change in osteoclast
pH(i) ... The
CSF-1 induced rise in
pH(i) is not blocked by 4,4'-diisothiocyanatostilbene-2,2'-disulfonic acid, an inhibitor of HCO ( 3 ) ( - ) transporters but is abolished by removing extracellular sodium
Rubin Kelley et al., Kidney Int Suppl 1994
(Lupus Nephritis...) :
These glomerular M
phi require
CSF-1 to survive and proliferate
Walker et al., Immunol Invest 1993
(Fibrosarcoma) :
T-cell reactivity against allogeneic MHC class II molecules was significantly suppressed by tumor bearing host ( TBH ) M
phi in the
presence of
M-CSF
Moore et al., J Immunol 1996
(Lupus Nephritis) :
We hypothesized that TNF-alpha enhanced the M
phi response to
CSF-1 in MRL-lpr mice
Moore et al., J Immunol 1996
(Lupus Nephritis) :
We used a gene transfer strategy to deliver CSF-1 into the kidney to evaluate M
phi response to
CSF-1 ...
CSF-1-TEC + BMM phi
caused a greater accumulation of M
phi in the implant site and interstitium of MRL +/+ than C3H +/+ mice ... We suggest that the response of MRL M phi to
CSF-1 is
responsible for the notable accumulation of M
phi in the MRL-lpr kidney