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APCS — HDAC11
Text-mined interactions from Literome
Sun et al., Genetics 1999
:
The effects on silencing at all three loci are dependent upon the
histone deacetylase activity of Rpd3. Enhanced silencing associated with sin3Delta, rpd3Delta, and
sap30Delta is differentially
dependent upon Sir2 and Sir4 at the telomeric and rDNA loci and is also dependent upon the ubiquitin conjugating enzyme Rad6 ( Ubc2 )
Villagra et al., Nat Immunol 2009
:
Overexpression of
HDAC11 inhibited IL-10 expression and
induced inflammatory
APCs that were able to prime naive T cells and restore the responsiveness of tolerant CD4+ T cells