Gene interactions and pathways from curated databases and text-mining

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CSF1 — JUN

Text-mined interactions from Literome

Hohensinner et al., Journal of thrombosis and haemostasis : JTH 2007 : To determine a possible role of nuclear factor kappaB (NF-kappaB) and activating protein 1 (AP-1) in M-CSF regulation, blockers to both pathways and an adenovirus overexpressing a dominant negative ( dn ) form of IkappaB kinase 2 (IKK2) were used
Rovida et al., J Immunol 2008 : Treatment of macrophages with ERK5-specific small interfering RNA markedly reduced CSF-1 induced DNA synthesis and total c-Jun phosphorylation and expression, while increasing the expression of the cyclin dependent kinase inhibitor p27
Chen et al., Hum Immunol 2008 : L-selectin ligation induced CSF-1 gene transcription is regulated by AP-1 in a c-Abl kinase dependent manner
Hwang et al., Arthritis Res Ther 2012 (Arthritis, Rheumatoid) : Importantly, the preferential induction of IL-34 rather than M-CSF by TNFa in RAFLS was mediated by the transcription factor nuclear factor kappa B ( NF-?B ) and activation of c-Jun N-terminal kinase (JNK)
Behre et al., J Biol Chem 1999 : We asked the question of whether c-Jun could promote the induction of the M-CSF receptor by collaborating with PU.1 ... The expression of this mutant form of c-Jun also completely blocks 12-O-tetradecanoylphorbol-13-acetate induced M-CSF receptor promoter activity during monocytic differentiation