Gene interactions and pathways from curated databases and text-mining

◀ Back to EGF

EGF — MUC5AC

Text-mined interactions from Literome

Takeyama et al., Proc Natl Acad Sci U S A 1999 (Hyperplasia) : Here we show that stimulation of epidermal growth factor receptors ( EGF-R ) by its ligands, EGF and transforming growth factor alpha ( TGFalpha ), causes MUC5AC expression in airway epithelial cells both in in vitro and in vivo
Mata et al., Thorax 2005 : Phosphodiesterase 4 inhibition decreases MUC5AC expression induced by epidermal growth factor in human airway epithelial cells ... Selective PDE4 inhibition is effective in decreasing EGF induced MUC5AC expression in human airway epithelial cells
Hegab et al., Chest 2007 (Bronchial Neoplasms...) : Niflumic acid also blocked the epidermal growth factor induced MUC5AC and mucin staining in the NCI-H292 cell line
Huang et al., Biochem Biophys Res Commun 2007 : Moreover, activation of both p38 and ERK is required for synergistic induction of MUC5AC by NTHi and EGF
Heo et al., Phytother Res 2009 : Genistein and curcumin suppress epidermal growth factor induced MUC5AC mucin production and gene expression from human airway epithelial cells ... This study investigated whether genistein and curcumin affect epidermal growth factor (EGF) induced MUC5AC mucin production and gene expression from human airway epithelial cells
Lee et al., Phytomedicine 2011 : Glycyrrhizin and carbenoxolone were found to inhibit the production of MUC5AC mucin protein induced by EGF or PMA, and both compounds also inhibited the expression of MUC5AC mucin gene induced by EGF or PMA
Cho et al., Phytother Res 2011 : suppress epidermal growth factor- and phorbol ester induced MUC5AC mucin production and gene expression from human airway epithelial cells ... Oleanolic acid and ursolic acid were found to inhibit the production of MUC5AC mucin protein induced by EGF and PMA, and both compounds also inhibited the expression of MUC5AC mucin gene induced by EGF and PMA
Kim et al., Phytother Res 2012 : Phorbol ester or epidermal growth-factor induced MUC5AC mucin gene expression and production from airway epithelial cells are inhibited by apigenin and wogonin
Nie et al., Eur J Pharmacol 2012 (MAP Kinase Signaling System) : The current study comprehensively determined the involvement of MAPKs/AP-1 and IKKs/I?B/NF-?B in epidermal growth factor (EGF) induced A549 cells, and sought to ascertain the signaling pathways of naringin imparted in suppression of EGF induced MUC5AC secretion
Harrop et al., Exp Lung Res 2013 : This effect was confirmed by using mtCC1-2 cells where addition of TGF-ß ( 2 ) reduced the ability of IL-13/EGF to induce Muc5ac promoter expression in wild-type cells ; however, this decrease was absent in mutant promoter transfected cells
Romano et al., Ital J Gastroenterol 1994 : We also evaluated the effect of TGF alpha and EGF on insoluble ( adherent ) gastric mucin and found that both compounds dose-dependently stimulated adherent mucin in a time frame consistent with their protective effect