Gene interactions and pathways from curated databases and text-mining

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SOX9 — TCF19

Text-mined interactions from Literome

Akiyama et al., Genes Dev 2004 : We hypothesize that the inhibitory activity of Sox9 is caused by its ability to compete with Tcf/Lef for binding to beta-catenin, followed by degradation of beta-catenin
Blache et al., J Cell Biol 2004 (Carcinoma...) : We provide in vitro and in vivo evidence that a bipartite beta-catenin/TCF4 transcription factor , the effector of the Wnt signaling pathway, is required for SOX9 expression in epithelial cells
Hwang et al., FEBS Lett 2005 : AP-1 activation suppressed the expression of Sox-9 , a major transcription factor regulating type II collagen expression
Bastide et al., J Cell Biol 2007 : Sox9 expression requires an active beta-catenin-Tcf complex, the transcriptional effector of the Wnt pathway
Passeron et al., Proc Natl Acad Sci U S A 2007 : During melanocyte development from the neural crest, SOX10 regulates microphthalmia associated transcription factor , which controls a set of genes critical for pigment cell development and pigmentation, including dopachrome tautomerase and tyrosinase ... In adult and neonatal melanocytes, SOX9 regulates microphthalmia associated transcription factor , dopachrome tautomerase, and tyrosinase promoters, leading to an increase in the expression of these key melanogenic proteins and finally to a stimulation of pigmentation
Chen et al., Liver Int 2013 (Carcinoma, Hepatocellular...) : In addition, SOX4 in HepG2 cell can enhance ß-catenin/TCF activity by increasing ß-catenin level
Stöckl et al., J Cell Sci 2013 : We found that the Sox9 activity level affects the expression of the key transcription factor in adipogenic differentiation, C/EBPß, and that cyclin D1 mediates the expression of the osteogenic marker osteocalcin in undifferentiated adult bone-marrow derived rat MSC