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CSF1 — SYK
Text-mined interactions from Literome
Ba et al., Cell Immunol 2005
:
Overexpression of wild typed non-receptor tyrosine kinase
Syk , but not kinase dead mutant of Syk,
promoted the upregulation of the transcriptional activity of
CSF-1 promoter caused by antibody engagement of PSGL-1
Zou et al., J Biol Chem 2009
:
Expression of nonphosphorylatable
Syk ( Y317F ) in primary Syk ( -/- ) OCs
enhances M-CSF- and alphavbeta3 induced phosphorylation of the cytoskeleton organizing molecules, SLP76, Vav3, and PLCgamma2, to levels greater than wild type, thereby accelerating the resorptive capacity of the cell