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AKAP3 — PRKAR1B

Text-mined interactions from Literome

Carr et al., Biochem J 1999 : These results suggest that altered PKA-AKAP interactions may contribute to the distinct responses of PA and PO follicles to high levels of cAMP, and that higher cAMP levels are required to activate PKA in PO ovaries
Sun et al., J Biol Chem 2000 : Phosphorylation reactions mediated by CFTR associated PKA II are inhibited by Ht31 peptide but not by the control peptide Ht31P, indicating that a protein kinase A anchoring protein ( AKAP ) is responsible for the association between PKA and CFTR
Zakhary et al., J Biol Chem 2000 : PKA stimulation of intact Chinese hamster ovary cells increased RIIalpha binding to AKAP100/mAKAP and AKAP15/18 by approximately 7- and 82-fold, respectively
Niu et al., Curr Biol 2001 : Interaction of heterotrimeric G13 protein with an A-kinase anchoring protein 110 (AKAP110) mediates cAMP independent PKA activation ... Finally, AKAP110 significantly potentiated G alpha 13-induced activation of PKA ... Finally, AKAP110 significantly potentiated G alpha 13-induced activation of PKA
Brown et al., Biol Reprod 2002 : By compartmentalizing PKA, AKAP140 and/or other AKAPs could spatially regulate PKA activity during oocyte development
Affaitati et al., J Biol Chem 2003 : Here we show that conditional expression of AKAP121 in PC12 cells selectively enhances cAMP.PKA signaling to mitochondria
Chen et al., Eur J Cell Biol 2006 : We have found that the functional regulation of the I ( Ks ) channel by PKA requires the A kinase anchoring protein ( AKAP ) Yotiao
Jeske et al., Pain 2009 (Calcium Signaling) : Recent studies have reported that the A-kinase Anchoring Protein 150 ( AKAP150 ) mediates PKA phosphorylation of TRPV1 in several nociceptive models
Christian et al., J Biol Chem 2011 (Chronic Disease...) : Small molecule AKAP-protein kinase A (PKA) interaction disruptors that activate PKA interfere with compartmentalized cAMP signaling in cardiac myocytes
Sekiguchi et al., Br J Pharmacol 2013 (Hyperalgesia) : In NG108-15 cells and/or rat DRG neurons, dibutyryl cAMP ( db-cAMP ) or PGE ( 2 ) increased T currents, an effect blocked by AKAP St-Ht31 inhibitor peptide ( AKAPI ) or KT5720, a PKA inhibitor
Dacher et al., J Neurosci 2013 : Using immunofluorescence and whole-cell patch-clamp recording in rat midbrain slices, we show that activation of postsynaptic D ( 2 ) -like family of dopamine ( DA ) receptor in the ventral tegmental area ( VTA ) induces long-term depression ( LTD ) of GABAergic synapses on DA neurons through an inositol triphosphate receptor mediated local rise in postsynaptic Ca ( 2+ ) and CaN activation accompanied by PKA inhibition, which requires AKAP150 as a bridging signaling molecule
Hillman et al., PloS one 2013 : Moreover, inhibition of PKA activity by H89, or its activation using 8Br-cAMP, increased AKAP3 degradation rate