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ANG — CA2

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Hirafuji et al., Eur J Pharmacol 1999 : Ni2+ ( 1 mM ) significantly inhibited the transient increase in intracellular Ca2+ concentration ([Ca2+]i) induced by Ang II ( 100 nM ) in aortic smooth muscle cells, as measured using fura-2
Sharma et al., J Lab Clin Med 2001 (Calcium Signaling) : ANG II prevented the accumulation of Ca2 ( + ) in intracellular stores ... In summary, ANG II increases extracellular/intracellular calcium dependent bidirectional Ca2 ( + ) transport in GECs, inhibits BK induced release of Ca2 ( + ) from IP ( 3 ) sensitive stores, and, in addition, reduces refilling of endoplasmic reticulum [ Ca2 ( + ) ] depleted by repeated BK stimulation
Hus-Citharel et al., Kidney Int 2002 : In the rat cortical thick ascending limb ( CTAL ), intracellular Ca2+ ( [ Ca2+ ] i ) responses to angiotensin II (Ang II) and angiotensin III (Ang III) were mediated by the Ang II subtype 1A receptor (AT1A-R), whereas the arginine vasopressin (AVP) dependent cAMP accumulation involved the vasopressin receptor type 2 ( V2-R )
Gerzanich et al., Hypertension 2003 (Hypertension) : These data provide the first evidence linking Ang induced eNOS mislocalization, eNOS dysfunction, and Ca2+ channel upregulation, and they provide novel mechanistic insights into pathological changes in LSAs associated with stroke
Pamidimukkala et al., Am J Physiol Heart Circ Physiol 2003 : In neurons preincubated with 100 nM E2, ANG II increased [ Ca2+ ] i by only 68 +/- 11 %, for a total inhibition of the ANG II-evoked response of 24 %
Zhu et al., Am J Physiol Heart Circ Physiol 2005 (Hypertension) : These results indicate that ANG II suppression secondary to elevated dietary salt intake impairs vascular relaxation and Ca2+ regulation by prostacyclin
Alvarez et al., Br J Pharmacol 2006 : In rat aortic smooth muscle cells ( RASMC ), EGCG ( 100 microM ) reduced increases in cytosolic free Ca2+ concentration ([Ca2+]i) induced by angiotensin II ( ANG II, 100 nM ) and KCl ( 60 mM ) in 1.5 mM CaCl2 -containing solution and by ANG II ( 100 nM ) in the absence of extracellular Ca2+
Han et al., J Huazhong Univ Sci Technolog Med Sci 2005 (Cardiomegaly) : The effects of cyclosporine A ( CsA ) on Angiontensin II (Ang II) induced protein contents, c-fos protein levels and cytosolic Ca2+ level ( [ Ca2+ ] i ) in cultured cardiomyocytes of neonatal rats were observed
Chobanian et al., Am J Physiol 1991 : When extracellular [ Na+ ] = intracellular [ Na+ ] ANG II significantly increased ammonia production in PCT. Absence of extracellular Ca2+ or addition of trifluoperazine or N- ( 6-aminohexyl ) -5-chloro-1-naphthalene sulfonamide ( W-7 ) ( Ca2 ( + ) -calmodulin dependent pathway inhibitors ) blocked the action of ANG II to enhance ammonia production
Chatsudthipong et al., Am J Physiol 1991 : Furthermore, the observation that TMB-8, an agent that prevents intracellular Ca2+ mobilization, completely eliminated the effect of ANG II strongly suggests that intracellular Ca2+ rather than Ca2+ influx mediates effects of ANG II on proximal tubular transport
Smith et al., J Membr Biol 1987 : The removal of external Ca2+ prevented A23187, but not ANG , from stimulating the cotransporter
Favre et al., Am J Hypertens 1988 : Angiotensin II (Ang II) and potassium ( K+ ) increase aldosterone ( Aldo ) production in vitro via Ca2+ dependent mechanisms
Takeda et al., Am J Physiol 1988 : Even in Ca2+-free medium, ANG II increased [ Ca2+ ] i and Ca2+ efflux, but to a lesser extent
Ding et al., Thromb Haemost 1985 : The effects of angiotensin II (ANG II) alone and in combination with other agonists on human platelet aggregation, thromboxane B2 ( TxB2 ) and cytosolic [Ca2+ ] i were investigated
Zhang et al., Hypertension 1996 : Angiotensin II (Ang II) and arginine vasopressin (AVP) increased intracellular free Ca2+ concentration [ Ca2+ ] i and/or the [ Ca2+]i transient rate (CaTR) in cultured neonatal rat cardiomyocytes
Wang et al., J Neurochem 1996 : Ang II increased [ Ca2+ ] i in 96.1 and 95.6 % of the polygonal and stellate glial cells, respectively
Consolim-Colombo et al., Am J Physiol 1996 : Because activation of AP neurons by either ANG II or AVP increases intracellular free Ca2+ concentrations ([Ca2+]i), the goal of this study was to analyze the factors affecting the [ Ca2+ ] i responses to ANG II and AVP
Côrtes et al., Am J Physiol 1996 (Hypertension) : The aim of the present work was to characterize angiotensin II (ANG II) receptors and their effect on intracellular free Ca2+ concentration ([Ca2+]i) in proliferating aortic smooth muscle cells ( VSMCs ) from spontaneously hypertensive rats ( SHR ) and normotensive Wistar-Kyoto rats ( WKY )
Zhu et al., Am J Physiol 1996 : ANG II ( 10 ( -7 ) M ) increased [ Ca2+ ] i from 50 to 240 nM in arteriolar and from 57 to 201 nM in aortic VSMC ( P < 0.001 for both )
Iversen et al., Am J Physiol 1998 : Angiotensin II (ANG II) and arginine vasopressin (AVP), at concentrations of 10 ( -10 ) -10 ( -6 ) M, produced dose dependent increases in [ Ca2+ ] i ; maximum increases were 221 +/- 49 nM for ANG II and 237 +/- 49 nM for AVP
Qi et al., Zhongguo Yao Li Xue Bao 1996 : Captopril ( Cap ) and enalapril ( Ena ) had no inhibitory effect on KCl-, NE-, and Ang induced [Ca2+ ] i increases in ASMC of WKY
Skolnick et al., Am J Physiol 1998 (Myocardial Infarction) : Effect of ANG II on pHi, [Ca2+ ] i, and contraction in rabbit ventricular myocytes from infarcted hearts
Szabadkai et al., Biochem Pharmacol 1999 : However, the mode of action of the two stimuli are different : while a dihidropyridine-sensitive mechanism contributes to the response to both agonists, Ang II induces Ca2+ release from intracellular stores and causes capacitative Ca2+ influx, whereas K+ was recently shown to activate a plasma membrane Ca2+ current ( Igl ) independently of membrane depolarization