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AR — GSK3B

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: GSK3B — AR (direct interaction, pull down) Wang et al., J Biol Chem 2004*
• IRef Biogrid Interaction: GSK3B — AR (physical association, affinity chromatography technology) Wang et al., J Biol Chem 2004*

Text-mined interactions from Literome

Salas et al., J Biol Chem 2004 (Prostatic Neoplasms) : Conversely, the glycogen synthase kinase-3 beta inhibitor lithium chloride suppressed the glycogen synthase kinase-3 beta mediated phosphorylation of the androgen receptor , thereby enabling androgen receptor-driven transcription to occur ... Because glycogen synthase kinase-3 beta activity is suppressed after the enzyme is phosphorylated by protein kinase B/Akt and Akt activity frequently increases during the progression of prostate cancer, nullification of the glycogen synthase kinase-3 beta mediated suppression of androgen receptor activity by Akt likely contributes to prostate cancer progression
Liao et al., Endocrinology 2004 (Prostatic Neoplasms) : However, the phosphatidylinositol 3kinase or GSK-3beta inhibitors did not block R1881 induced nuclear translocation of androgen receptor