Gene interactions and pathways from curated databases and text-mining

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BCL6 — MAPK1

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Quadros et al., Cancer Biol Ther 2006 (Carcinoma, Squamous Cell) : This effect is due, in part, to sustained MAPK dependent Bcl-xL expression
Alfano et al., J Biol Chem 2006 : Accordingly, incubation of retinal pigment epithelial cells with uPA elicits a time dependent enhancement of MAPK and phosphatidylinositol 3-kinase activities as well as the transcriptional activation of Bcl-xL anti-apoptotic factor
Batlle et al., Mol Immunol 2009 (Blast Crisis...) : We show that p38 MAPK mediates induction of Bcl-6 following inhibition of Bcr-Abl by imatinib
Chen et al., Toxicology 2009 (Neuroblastoma) : SB202190 ( p38 MAPK inhibitor ) and SP600125 ( JNK inhibitor ) attenuated mitochondrial depolarization, degradation of Bcl-2/Bcl-xL , and mitochondrial translocation of Bax
Chen et al., J Cell Physiol 2010 (Neuroblastoma) : Moreover, phospho-p38 MAPK and phospho-JNK were proved to be involved in Bcl-xL degradation, and overexpression of Bcl-xL attenuated the cytotoxic effect of notexin
Zhao et al., Brain Res 2013 (Brain Edema...) : In addition, we found SB203580 significantly decreased P-p38 MAPK levels, and inhibited HPC induced mitochondria translocation of Bcl-xL in the brain of HPC and MCAO treated mice