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CA2 — JUN

Text-mined interactions from Literome

Crossthwaite et al., J Neurochem 2004 : Demonstrating that PI3-kinase is not ubiquitously involved in NMDA receptor signal transduction, the PI 3-kinase inhibitors wortmannin and LY294002 did not prevent NMDA receptor Ca2+ dependent phosphorylation of c-Jun N-terminal kinase 1/2 ( JNK1/2 )
Wang et al., Clin Cancer Res 2006 (Colonic Neoplasms) : This stimulation involves Ca2+ dependent protein kinase C, extracellular signal regulated kinase dependent activator protein-1 , and extracellular signal regulated kinase independent nuclear factor-kappaB pathways
Wu et al., Glia 2009 : IL-1beta induces MMP-9 expression via a Ca2+ dependent CaMKII/JNK/c-JUN cascade in rat brain astrocytes
Vandenplas et al., Biochem J 1990 : Increased intracellular Ca2+ is necessary for maximal expression of the proto-oncogene c-jun in the Jurkat T-cell line
Garcia-Sanchez et al., Mol Cell Biochem 2012 (MAP Kinase Signaling System...) : Resveratrol mobilizes Ca2+ from intracellular stores and induces c-Jun N-terminal kinase activation in tumoral AR42J cells
Li et al., J Biol Chem 2013 (MAP Kinase Signaling System) : Wingless-type Mammary Tumor Virus Integration Site Family, Member 5A ( Wnt5a ) Regulates Human Immunodeficiency Virus Type 1 ( HIV-1 ) Envelope Glycoprotein 120 (gp120) induced Expression of Pro-Inflammatory Cytokines via the Ca2+/Calmodulin dependent Protein Kinase II ( CaMKII ) and c-Jun N-terminal Kinase (JNK) Signaling Pathways
Esteve et al., Neuropharmacology 1995 : The cytosolic calcium rise and AP1 binding activation elicited by haloperidol were dependent on extracellular Ca2+ , suggesting that haloperidol exerted its effects by promoting Ca2+ entry into PC12 cells
Biswas et al., Phytochemistry 1996 : The activity of AP-I was not affected by 1 mM Mg2+, Mn2+, Ca2+ , Co2+ or Pb2+, but severely inhibited by 1 mM Cu2+, Fe3+, Hg2+, Mo6+ or Zn2+