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CA2 — TNFSF11

Text-mined interactions from Literome

Shin et al., Exp Mol Med 2003 : High Ca2+ ( e ) alone continuously up-regulated RANKL expression while only transiently increased OPG expression
Xiao et al., Zhonghua Yi Xue Za Zhi 2004 (Bone Resorption) : RANKL plus AA significantly down-regulated the mRNA expression of CAII and RANK by 60 % and 20 % respectively
Seales et al., J Cell Biochem 2006 (Bone Resorption...) : During osteoclastogenesis, RANKL-induction stimulates a rise in intracellular Ca2+ , which in turn activates calmodulin and its downstream effectors
Fujisaki et al., Life Sci 2007 : These results indicate that the expression of CAII , cathepsin K, and MMP-9 in RAW264.7 cells is not induced by M-CSF, but by RANKL in the presence of IL-1alpha
Kim et al., Mol Nutr Food Res 2012 (Bone Resorption) : The phloretin treatment retarded RANKL induced expression of carbonic anhydrase II , vacuolar-type H ( + ) -ATPase D2 and ß3 integrin, all involved in the bone resorption
Zhang et al., Biochimie 2012 (Bone Resorption) : Besides, RANKL induced osteoclast formation from RAW 264.7 cells and the expression of TRAP, CA II , CTSK and MMP-9 was significantly reduced by the treatment of 25 µM HEF and 17ß-estradiol ( ES ), and the inhibitory strength increases in the order TF < ES < ICA < BS, which was blocked by ICI182780 suggesting that the regulation of osteoclast activity might be ER dependent
Kim et al., Am J Chin Med 2012 : In addition, the YFIE treatment retarded RANKL induced cathepsin K production and carbonic anhydrase II expression, both of which are involved in bone resorption