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CPOX — IL10

Text-mined interactions from Literome

Ramaswamy et al., J Immunol 2000 (Hypersensitivity, Delayed...) : COX2 inhibitors blocked the parasite induced PGE ( 2 ) and IL-10 from keratinocytes
Moore et al., J Appl Physiol 2001 : In human cultured airway smooth muscle cells, interleukin (IL)-1 beta increases cyclooxygenase (COX)-2 expression and PGE ( 2 ) release, ultimately resulting in decreased beta-adrenergic responsiveness
Williams et al., J Immunol 2004 : STAT3 DN also blocked IL-10 mediated inhibition of MHC class II and COX2 expression
Petit-Bertron et al., Cytokine 2005 : The discovery that IL-10 can inhibit the production of coproporphyrinogen oxidase ( an enzyme involved in the synthesis of heme ) may shed some lights on the mechanisms of anaemia induced by IL-10
Helmersson et al., Free radical research 2005 (Cardiovascular Diseases...) : This study investigated the longitudinal association between serum selenium ( s-Se ) and a golden standard indicator of oxidative stress in vivo ( 8-iso-prostaglandin F2alpha, a major F2-isoprostane ), an indicator of cyclooxygenase (COX) mediated inflammation ( prostaglandin F2alpha ), high sensitive C-reactive protein ( hsCRP ), interleukin-6 (IL-6) and serum amyloid A protein (SAA) in a follow-up study of 27 years
Li et al., J Infect Dis 2006 (Disease Models, Animal...) : Tumor necrosis factor (TNF)-alpha and interleukin-10 mRNA expression was elevated in H. pylori infected mice, but only TNF-alpha mRNA expression was further increased by COX deficiency
Carregaro et al., J Leukoc Biol 2008 (Inflammation...) : COX inhibition resulted in diminished SGE induced IL-10 production, and PGE2 release triggered by SGE remained increased in IL-10-/- mice, suggesting that prostanoids are acting through an IL-10 dependent mechanism
Inada et al., J Immunotoxicol 2011 : Endogenous COX inhibition with propofol, as well as with the selective COX-2 inhibitor, NS-398, did not affect IL-12 and IL-10 production from DC
Obermajer et al., Transplantation research 2012 : We observed that PGE2 induces endogenous cyclooxygenase (COX)2 expression in cultured monocytes, blocking their differentiation into CD1a+ dendritic cells (DCs) and inducing the expression of indoleamine 2,3-dioxygenase 1, IL-4Ra, nitric oxide synthase 2 and IL-10 - typical MDSC associated suppressive factors
Niiro et al., Blood 1997 : Moreover, IL-10 and IL-4 inhibited LPS induced cyclooxygenase (COX) activity in neutrophils
Endo et al., Am J Respir Cell Mol Biol 1998 : Lipopolysaccharide (LPS) stimulated PGE2 synthesis and COX activity were suppressed significantly by IL-4, but were not affected significantly by IL-10 , IL-13, or IFN-gamma
Dean et al., J Biol Chem 1999 : We have previously shown that the pyridinyl imidazole SB 203580, which inhibits it, blocks the interleukin-1 induction of cyclooxygenase-2 (COX-2) and matrix metalloproteinase 1 and 3 mRNAs in fibroblasts