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FES — KIT

Pathways - manually collected, often from reviews:

• Reactome Reaction: FES → KIT (indirect_complex) Masuhara et al., Biochem Biophys Res Commun 2000*, Craig et al., Mol Cell Biol 2002*, Roskoski et al., Biochem Biophys Res Commun 2005, Tang et al., Mol Cell Biol 1994*, Jhun et al., J Biol Chem 1995*, Price et al., J Biol Chem 1997
• Reactome Reaction: FES → KIT (reaction) Masuhara et al., Biochem Biophys Res Commun 2000*, Craig et al., Mol Cell Biol 2002*, Roskoski et al., Biochem Biophys Res Commun 2005, Tang et al., Mol Cell Biol 1994*, Jhun et al., J Biol Chem 1995*, Price et al., J Biol Chem 1997

Text-mined interactions from Literome

Voisset et al., Blood 2007 : FES is phosphorylated on tyrosine residues in cells that carry KIT ( D816V ) mutation, and this phosphorylation is KIT dependent
Voisset et al., Leukemia 2010 (Leukemia, Myeloid, Acute) : We have recently shown that FES but not FER is necessary for oncogenic KIT receptor signaling
Voisset et al., Biochem Biophys Res Commun 2010 : Unlike in the context of oncogenic KIT mutant, FES is not involved in wild-type KIT proliferation signal, or in cell adhesion