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GSK3B — IGF2

Text-mined interactions from Literome

Sinha et al., American journal of physiology. Renal physiology 2005 : Taken together, these data suggest that Li ( + ) or BIO promotes renal epithelial cell survival by inhibiting apoptosis through GSK3beta dependent activation of the Wnt pathway and subsequent release of IGF-II
Sjostrom et al., Dev Cell 2005 (Neuroblastoma) : In proliferating cerebellar granule neuron precursors ( CGNPs ), Sonic hedgehog signaling induces N-myc expression, and N-myc protein is stabilized by insulin-like growth factor mediated suppression of GSK-3beta
Carter et al., Acta Neuropathol 2008 (Brain Neoplasms...) : Aspartyl (asparaginyl)-beta-hydroxylase ( AAH ) is a mediator of neuronal motility, and stimulated by insulin and IGF activation of PI3 kinase-Akt, or inhibition of GSK-3beta