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IKBKG — NOX1

Text-mined interactions from Literome

Wu et al., Free Radic Biol Med 2009 (Inflammation...) : Collectively, the results of this study and those of previous reports indicate that the conversion of cardiomyocytes to a proinflammatory phenotype in sepsis involves two distinct pathways : NADPH oxidase mediated phosphorylation of IKK and p38 MAP kinase mediated phosphorylation of p65
Menden et al., Am J Physiol Lung Cell Mol Physiol 2013 : LPS mediated endothelial activation in pulmonary endothelial cells : role of Nox2 dependent IKK-ß phosphorylation ... We examined whether Nox2 dependent, LPS mediated IKK-ß phosphorylation was regulated by protein phosphatase 2A (PP2A) or TGF-ß associated kinase-1 (TAK1) in HPMEC
Valente et al., Free Radic Biol Med 2013 : Advanced oxidation protein products induce cardiomyocyte death via Nox2/Rac1/superoxide dependent TRAF3IP2/JNK signaling ... AOPP-MSA stimulated Nox2/Rac1 dependent superoxide generation, TRAF3IP2 expression, and TRAF3IP2 dependent JNK activation ... These results demonstrate for the first time that AOPPs induce cardiomyocyte death via Nox2/Rac1/superoxide dependent TRAF3IP2/JNK activation in vitro and suggest that AOPPs may contribute to myocardial injury in vivo
Valente et al., Cell Signal 2013 : IL-18 induced Nox1 dependent ROS generation, TRAF3IP2 expression, and IKK/NF-?B and JNK/AP-1 activation