Gene interactions and pathways from curated databases and text-mining

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CAT — IL1A

Text-mined interactions from Literome

Osawa et al., J Biochem 2000 : However, IL-1 did not stimulate the transcription of a CAT-reporter gene having two copies of the p53-responsive element on its promoter or the p53 binding capacity of nuclear extracts, although it increased transcriptional rate of p21 ( WAF1 ) in these high-passage cells
Wang et al., Mol Psychiatry 2004 : Further confirming the role of p38 pathways, pretreatment of LMCAT cells with antisense oligonucleotides targeted to p38 MAPK completely abrogated IL-1alpha inhibition of Dex induced GRE-CAT activity
Baldari et al., J Biol Chem 1991 : IL-1 in combination with PHA but not with PMA resulted in induction of CAT activity in these cells
Mathy-Hartert et al., Osteoarthritis Cartilage 2008 : This work was designed to assess the effects of interleukin (IL)-1beta and IL-6 on the enzymatic activity and gene expression of SODs, CAT and GPX in bovine chondrocytes
Donati et al., J Immunol 1990 : Protein kinase inhibitors H7 ( 5-50 microM ) and sphingosine ( 50 microM ) inhibited only IL-1 induced CAT activity, whereas H8 ( 5-50 microM ) and HA1004 ( 50 microM ) were ineffective on both parameters ... PMA ( 0.5 microM ) and R59 022, a diacylglycerol kinase inhibitor ( 10 microM ), did not modify either control or IL-1 induced CAT activity
Kobayashi et al., Biochem Biophys Res Commun 1989 : Among the cytokines tested here ( IL-2, IL-3, IL-4, IL-5, IL-6, granulocyte colony stimulating factor ( G-CSF ), granulocyte/macrophage colony stimulating factor ( GM-CSF ), interferon-alpha ( IFN-alpha ), interferon-beta (IFN-beta) and interferon-gamma ( IFN-gamma ] only interleukin 1(IL-1) augmented HIV-long terminal repeat ( LTR ) directed chloramphenicol acetyl transferase(CAT) activity in protein kinase C ( PKC ) -independent manner
Leff et al., Am J Physiol 1995 : IL-1 induced tolerance to IL-1 mediated lung leak and the associated increases in lung catalase , lung G6PDH, and serum catalase activities were all prevented by treating rats with the IL-1-receptor antagonist or with N-acetyl-L-cysteine, an agent that increases intracellular glutathione levels
Oliveira et al., Mol Cell Biol 1994 : Inhibition of TNF- or IL-1 induced CAT activity by IFN-beta or IFN-alpha was also observed when a DNA fragment containing only the NF-IL-6 and NF-kappa B sites ( positions -94 to -70 ) was placed upstream of the homologous or a heterologous minimal promoter
Derevianko et al., Shock 1998 : IL-1betaR type I expression was significantly up-regulated in the presence of catalase and SOD