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IL1A — TCF25

Text-mined interactions from Literome

Tamaoki et al., Am J Med 2004 (Chronic Disease...) : Macrolides inhibit the production of many proinflammatory cytokines, such as interleukin (IL)-1 , IL-6, IL-8, and tumor necrosis factor-alpha, perhaps by suppressing the transcription factor nuclear factor-kappaB or activator protein-1
Ali et al., Int J Biochem Cell Biol 2010 : IL-1 induced NF-kappaB DNA binding and activation by this transcription factor and this was attenuated by curcumin and apigenin
Bomsztyk et al., J Biol Chem 1990 : IL-1 alpha transiently activated NF-kappa B transcription factor
Montaseri et al., PloS one 2011 : Herein, we investigated the potential synergistic effects of insulin-like growth factor ( IGF-1 ) and platelet derived growth factor ( PDGF-bb ) on different mechanisms participating in IL-1ß induced activation of nuclear transcription factor-?B ( NF-?B ) and apoptosis in chondrocytes
Tong et al., J Neurosci 2012 : Using rat organotypic hippocampal cultures, we found that IL-1ß suppressed BDNF dependent regulation of Arc and phosphorylation of cofilin and cAMP response element binding protein ( CREB ), a transcription factor regulating Arc expression
Lee et al., J Biol Chem 1994 : In addition, both TPA and IL-1 alpha caused increases not only in the phosphorylation of c-Jun and c-Fos protein but also in the transactivating activity of AP-1 nuclear transcription factor
Jimi et al., J Biol Chem 1998 : We previously reported that interleukin-1 (IL-1) promoted the survival of murine osteoclast-like cells ( OCLs ) formed in vitro and activated a transcription factor , NF-kappaB, of OCLs