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JUN — NPY6R

Text-mined interactions from Literome

Al-Murrani et al., Biochem J 1999 : Expression of I2PP2A also increased the transcriptional activity of activator protein-1 , and this effect was diminished in a dose dependent manner by expression of PP2AC ... Densitometric analysis following Western blotting of extracts with antibodies specific for phospho-Ser63 and Ser73 suggests that the effects of I2PP2A and PP2AC expression might be mediated , in part, by changes in the phosphorylation of c-Jun at Ser63
Smolinska et al., Mol Immunol 2008 : In contrast, PP2 did inhibit AP-1 nuclear accumulation in response to LPS
Wu et al., Br J Pharmacol 2009 : Effects of tripterine were investigated on endothelial barrier function, inducible nitric oxide synthase (iNOS) expression, nicotinamide adenine dinucleotide phasphate ( NADPH ) oxidase activity, 3-nitrotyrosine formation, protein phosphatase type 2A (PP2A) activity, activation of extracellular regulated kinase (ERK), c-Jun terminal kinase (JNK) and Janus kinase ( Jak2 ), and degradation of IkappaB in microvascular endothelial cells exposed to pro-inflammatory stimulus [ lipopolysaccharide (LPS) + interferon gamma (IFNgamma) ] and on vascular permeability in air pouches of mice injected with LPS + IFNgamma
Ronda et al., J Steroid Biochem Mol Biol 2010 : Accordingly, PP2 and Ro318220 suppressed CREB and Elk-1 phosphorylation as well as c-Fos and c-Jun oncoprotein levels induced by 17ß-estradiol
Tang et al., Arthritis Rheum 2010 (Osteoarthritis) : CCL5 mediated AP-1 luciferase activity and c-Jun binding to the AP-1 element were inhibited by Met-RANTES, rottlerin, and PP2
Chen et al., PloS one 2012 : HGF mediated AP-1-luciferase activity and c-Jun binding to the AP-1 element was reduced by c-Met inhibitor, Ly294002, Akt inhibitor, and PP2