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MIP — NFKB1

Text-mined interactions from Literome

Dejardin et al., Immunity 2002 : IKKbeta dependent activation of canonical NF-kappaB is required for the expression but not processing of p100 and for the expression of proinflammatory molecules including VCAM-1, MIP-1beta, and MIP-2 in response to LTbetaR ligation
Kwon et al., Biochem Biophys Res Commun 2003 : Inhibition of NF-kappaB nuclear localization by coexpression of a mutant IkappaBalpha protein blocked CpG-ODN induced transcription from a MIP-2 promoter-reporter construct, showing that NF-kappaB activation is required for MIP-2 gene expression in the CpG-ODN signaling pathway
Hehlgans et al., Eur Cytokine Netw 2003 (Fibrosarcoma) : Activation of the lymphotoxin-beta receptor induces NFkappaB dependent interleukin-6 and MIP-2 secretion in mouse fibrosarcoma cells
Darieva et al., J Leukoc Biol 2004 (Inflammation...) : Specific pharmacological inhibition of the PI-3K, c-jun-N-terminal kinase ( JNK ), and to a smaller extent, p38 MAPK but not extracellular regulated kinase (ERK), suppressed NF-kappaB dependent reporter gene transcription and MIP-2 and NO secretion in BCG induced RAW264.7 macrophages
Kato et al., J Am Soc Nephrol 2004 (Peritonitis) : As compared with MCP-1, MIP-2 mRNA expression was inhibited by a high dose of curcumin but not by NF-kappaB decoy oligodeoxynucleotide and individual inhibitions of MAP kinase, suggesting that the additional signaling pathway with NF-kappaB might be involved in mRNA expression of MIP-2 ... These show that TLR4 is directly involved in the production of MCP-1 and MIP-2 by MPMC in a NF-kappaB dependent manner, but the process does not require any MAP kinase activation
Wang et al., J Immunol 2005 (Disease Models, Animal...) : Taken together, these results suggest that chronic morphine treatment in an S. pneumoniae infection model suppresses NF-kappaB gene transcription in lung resident cells, which, in turn, modulates the transcriptional regulation of MIP-2 and inflammatory cytokines
Sadikot et al., J Immunol 2006 (Cross Infection...) : In contrast, NF-kappaB inhibition reduced MIP-2 expression and neutrophil influx following P. aeruginosa infection
Kim et al., Mol Cells 2008 : In addition, rosmarinic acid reduced cell migration by inducing the expression of a specific chemokine receptor on LPS induced mature BMDCs. Rosmarinic acid also significantly reduced the expressions of MCP-1 and MIP-lalpha induced by LPS in BMDCs and inhibited LPS induced activation of MAPK and the nuclear translocation of NF-kappaB