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EPHB2 — TGFBR2

Text-mined interactions from Literome

Chin et al., American journal of physiology. Renal physiology 2001 : Moreover, dominant negative TbetaR-II ( M ) failed to block TGF-beta(1) stimulated pro-alpha ( 1 ) ( I ) collagen mRNA expression and cellular protein synthesis, whereas TGF-beta(1) induced extracellular signal regulated kinase ( ERK ) 1/ERK2 activation and antiproliferative responses were blocked by TbetaR-II ( M )
Huo et al., Cell Biol Toxicol 2007 (MAP Kinase Signaling System) : These data revealed that TbetaRII and Smad7 play the critical roles in TGF-beta1 mediated activation of ERK ; Smad3 and Smad4 can play an indirect role through up-regulating Smad7 expression ; and TGF-beta1 induced phosphorylation of ERK may participate in BEP2D cell proliferation and apoptosis regulation