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Akt → insulin: " The effects of RBP4 on nitric oxide ( NO ) and insulin stimulated endothelin-1 (ET-1) secretion and on phosphorylation ( p ) of Akt , endothelial NO synthetase (eNOS), and extracellular signal regulated kinase ( ERK ) 1/2 were investigated in bovine vascular aortic endothelial cells ( BAECs ) "
endothelial NO synthetase (eNOS) → insulin: " The effects of RBP4 on nitric oxide ( NO ) and insulin stimulated endothelin-1 (ET-1) secretion and on phosphorylation ( p ) of Akt, endothelial NO synthetase (eNOS) , and extracellular signal regulated kinase ( ERK ) 1/2 were investigated in bovine vascular aortic endothelial cells ( BAECs ) "
endothelin-1 (ET-1) — RBP4: " The effects of RBP4 on nitric oxide ( NO ) and insulin stimulated endothelin-1 (ET-1) secretion and on phosphorylation ( p ) of Akt, endothelial NO synthetase (eNOS), and extracellular signal regulated kinase ( ERK ) 1/2 were investigated in bovine vascular aortic endothelial cells ( BAECs ) "
Akt — RBP4: " The effects of RBP4 on nitric oxide ( NO ) and insulin stimulated endothelin-1 (ET-1) secretion and on phosphorylation ( p ) of Akt , endothelial NO synthetase (eNOS), and extracellular signal regulated kinase ( ERK ) 1/2 were investigated in bovine vascular aortic endothelial cells ( BAECs ) "
endothelial NO synthetase (eNOS) — RBP4: " The effects of RBP4 on nitric oxide ( NO ) and insulin stimulated endothelin-1 (ET-1) secretion and on phosphorylation ( p ) of Akt, endothelial NO synthetase (eNOS) , and extracellular signal regulated kinase ( ERK ) 1/2 were investigated in bovine vascular aortic endothelial cells ( BAECs ) "
p-ERK1/2 ⊣ p-Akt: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-ERK1/2 ⊣ p-eNOS: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-ERK1/2 ⊣ RBP4: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-ERK1/2 ⊣ p-Akt: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-ERK1/2 ⊣ p-eNOS: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-ERK1/2 ⊣ RBP4: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-Akt → RBP4: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-eNOS → RBP4: " RBP4 significantly increased p-Akt and p-eNOS production, and significantly inhibited p-ERK1/2 production "
p-Akt → RBP4: " Triciribine, an Akt inhibitor, and wortmannin significantly inhibited RBP4 induced p-Akt and p-eNOS production "
p-eNOS → RBP4: " Triciribine, an Akt inhibitor, and wortmannin significantly inhibited RBP4 induced p-Akt and p-eNOS production "