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CALM1 — PRKACG
Text-mined interactions from Literome
Galisteo et al., J Pharmacol Exp Ther 1999
:
Taken together, our results suggest that CYP3A induction by PB is regulated positively by
calmodulin dependent protein kinase and cGMP dependent protein kinase, and negatively by
PKA in mouse hepatocytes in primary culture
Tzavara et al., Eur J Neurosci 2000
(Substance Withdrawal Syndrome) :
We show that after SR141716A precipitation of cannabinoid withdrawal, basal and
calcium-calmodulin stimulated adenylyl cyclase activities as well as active
PKA in the cerebellum increase in a transient manner with a temporal profile which matches that of the somatic expression of abstinence
Tang et al., Acta Pharmacol Sin 2000
:
To study the effects of dopamine ( DA ) and glutamate ( Glu ) and their receptor agonists/antagonists on
Ca2+/calmodulin dependent protein kinase II ( CCDPK II ), cyclic AMP dependent
protein kinase A (PKA) activities and the LDH release in rat striatal slices, and to examine the interaction between DA and Glu transmitter systems in striatum
Kudoh et al., Brain Res 2001
(Magnesium Deficiency...) :
In this paper, protein kinase C ( PKC ) and
calcium/calmodulin dependent protein kinase type II ( CaMKII ) but not
protein kinase A (PKA) were reported to contribute to the potentiation mechanism during a step between the activation of the N-methyl-D-aspartate receptors by the Mg ( 2+ ) -free treatment and the secretion of the protein factor ( s )
Nesher et al., Diabetes 2002
(Diabetes Mellitus, Type 2) :
We suggest that three families of PKs (
calmodulin dependent PK [ CaMK ],
PKA , and PKC ) function as distal amplifiers for stimulus-secretion coupling signals originating from fuel metabolism, as well as from incretins acting through membrane receptors, adenylate cyclase, and phospholipase C
Kawaguchi et al., J Neurosci 2002
:
We confirmed that inhibition of Ca ( 2+ )
/calmodulin dependent protein kinase II ( CaMKII ) or
PKA suppresses RP
Kotak et al., J Neurobiol 2002
:
In this study, we asked whether the calcium dependency of inhibitory depression was associated with the activation of
calcium/calmodulin dependent protein kinase II ( CaMKII ), protein kinase C ( PKC ), and/or cAMP dependent
protein kinase A (PKA)
Yang et al., J Neurophysiol 2004
:
Considerable evidence has shown that
calcium/calmodulin dependent protein kinase II ( CaMKII ),
protein kinase A (PKA) , and protein kinase C ( PKC ) are important for LTP in hippocampus
Ciarimboli et al., J Cell Physiol 2004
:
hOCT1 expressed in CHO-cells was inhibited by
protein kinase A (PKA) activation ( 1 microM forskolin, -58 +/- 6 %, n = 12 ),
calmodulin inhibition ( 0.1 microM calmidazolium, -68 +/- 3 %, n = 6 ; 10 microM ophiobolin A, -48 +/- 10 %, n = 7 ), calmodulin dependent kinase II inhibition ( 1 microM KN62, -78 +/- 4 %, n = 12 ), and inhibition of p56 ( lck ) tyrosine kinase ( 10 microM aminogenistein, -35 +/- 7 %, n = 12 )
Hongpaisan et al., J Neurosci 2004
(Calcium Signaling) :
Large increases in cytosolic free Ca2+ ( [ Ca2+ ] i ) activate several kinases that are important for neuronal plasticity, including
Ca2+/calmodulin dependent kinase II ( CaMKII ),
protein kinase A (PKA) , and protein kinase C ( PKC )
Zheng et al., Pharmacol Ther 2005
(Heart Failure) :
More importantly, persistent beta1AR stimulation evokes a multitude of cardiac toxic effects, including myocyte apoptosis and hypertrophy, via a
calmodulin dependent protein kinase II ( CaMKII ) -, rather than
cAMP-PKA- , dependent mechanism in rodent heart in vivo and cultured cardiomyocytes
Huang et al., J Neurosci 2006
:
These Ng-/- mice, as compared with Ng+/+, respond poorly after treatment of their hippocampal slices with agents that activate signaling molecules important for learning and memory, including
Ca2+/calmodulin dependent protein kinase II ( alphaCaMKII ), PKC,
protein kinase A (PKA) , extracellular signal regulated kinase ( ERK ), and cAMP response element binding protein ( CREB )
Siarey et al., J Neurochem 2006
(Down Syndrome) :
Here we report that
calcium/calmodulin dependent protein kinase II ( CaMKII ), phosphatidylinositol 3-kinase (PI3K)/Akt, extracellular signal regulated kinase ( ERK ),
protein kinase A (PKA) and protein kinase C ( PKC ), all of which have been shown to be involved in synaptic plasticity, are altered in the Ts65Dn hippocampus
D'Angiolella et al., PloS one 2007
:
MPF down-regulation required Ca ( ++ )
/calmodulin dependent kinase II ( CaMKII ) and cAMP dependent
protein kinase (PKA) activities at meiosis and mitosis exit, respectively
Dash et al., Learn Mem 2007
(Memory Disorders) :
Additionally, the negative influence of calcium- and cAMP dependent protein kinase ( i.e.,
calcium/calmodulin dependent protein kinase II ( CaMKII ), calcium/diacylglycerol activated protein kinase C ( PKC ), and cAMP dependent
protein kinase A (PKA) ) activities on working memory will be reviewed
McConville et al., Am J Physiol Endocrinol Metab 2007
:
These findings are consistent with beta ( 1 ) -AR-specific
PKA dependent glycogen
phosphorylase kinase signaling
Machado et al., J Neurosci Res 2008
(MAP Kinase Signaling System) :
Inhibiting
calcium/calmodulin dependent kinase II ( CaMKII ), calcineurin, or
protein kinase A (PKA) activity prevents KCl induced IPD-IEG mRNA accumulation, suggesting that the IPD-IEG genes are induced by depolarization in neurons via a combination of calcineurin/PKA- and CaMKII dependent pathways
Boudreau et al., J Neurochem 2009
(Cocaine-Related Disorders...) :
We focused on
calcium/calmodulin dependent protein kinase II ( CaMKII ), extracellular signal regulated protein kinase ( ERK ), and
protein kinase A (PKA)
Ogrodnik et al., J Physiol 2010
(Calcium Signaling) :
This was likely to have been mediated by Ca ( 2+ )
/calmodulin dependent protein kinase ( CaMKII ), rather than cAMP dependent
protein kinase (PKA)
Song et al., J Biol Chem 2010
:
We found that Ser-21/9 phosphorylation of GSK-3 was mediated by Ca ( 2+ )
/calmodulin dependent protein kinase II ( CaMKII ) but not by Akt/PKB,
PKA , or p90 ( RSK )
Soltis et al., Biophys J 2010
(Arrhythmias, Cardiac...) :
Cardiac excitation-contraction coupling is a highly coordinated process that is controlled by protein kinase signaling pathways, including Ca ( 2+ )
/calmodulin dependent protein kinase II ( CaMKII ) and
protein kinase A (PKA) ... To better understand mechanisms of cardiac CaMKII function, we integrated dynamic CaMKII dependent regulation of key Ca ( 2+ ) handling targets with previously validated models of cardiac EC coupling, Ca ( 2+ )
/calmodulin dependent
activation of CaMKII, and ß-adrenergic activation of
PKA
McArdle et al., J Virol 2011
:
We find that inhibition of calmodulin dependent kinase kinase ( CaMKK ), but not
calmodulin dependent kinase II ( CaMKII ) or
protein kinase A (PKA) , blocks HCMV mediated activation of glycolysis
Moncada et al., Proc Natl Acad Sci U S A 2011
:
Moreover, activation of the glutamatergic NMDA receptors is required for setting the learning tags, and this machinery further required a-Ca ( 2+ )
/calmodulin dependent protein kinase II and
PKA but not ERK1/2 activity
Chiluiza et al., J Biol Chem 2013
(Glomerulosclerosis, Focal Segmental) :
However, these effects appear to be independent of their effects on
calcium/calmodulin dependent protein kinase II and
PKA , respectively
Tan et al., J Neurosci 1994
:
In this study 32P labeling of non-NMDA GluRs was investigated in cultured hippocampal neurons stimulated 2-15 min with agonists that selectively stimulate either
Ca2+/calmodulin dependent protein kinase II ( CaM-kinase II ), Ca2+/phospholipid dependent protein kinase C ( PKC ), or cAMP dependent
protein kinase A (PKA)
Menez et al., Alcohol Alcohol 1993
:
Phosphorylation of pure cytochrome P4502E1 (CYP2E1) was achieved in vitro using
Ca2+/calmodulin dependent protein kinase II ( CaM kinase II ), protein kinase C ( PKC ) and cAMP dependent
protein kinase (PKA)