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MET — PI3
Text-mined interactions from Literome
Nakanishi et al., Clin Exp Metastasis 1999
(Carcinoma, Hepatocellular) :
HGF stimulated the motility of Hep3B, HepG2, and Huh-7 cells in a dose dependent manner in association with tyrosine phosphorylation of
c-Met and
activation of
phosphatidylinositol 3-kinase (PI3-K)
Basar et al., Infect Immun 2005
:
Binding of InlB leads to phosphorylation of
Met and the adapter Gab1 and to
activation of host
phosphoinositide (PI) 3-kinase
Grotegut et al., Int J Oncol 2010
(Hepatoblastoma...) :
Here, we demonstrate that HGF has no impact on overall cell viability and proliferation of HB cells, although signal transduction occurs downstream of HGF, such as
c-Met phosphorylation,
activation of
phosphoinositide 3-kinase (PI3K)/AKT and mitogen activated protein kinase ( MAPK ) /ERK-1/2 signaling