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AHSA1 — CCL2

Text-mined interactions from Literome

Ashida et al., J Biol Chem 2001 : Further, C3 exoenzyme and the Rho kinase inhibitor blocked MCP-1 dependent p38-MAPK activation
Waterhouse et al., J Interferon Cytokine Res 2001 : Monocyte chemoattractant protein-1 production by intestinal epithelial cells in vitro : a role for p38 in epithelial chemokine expression
Tsuboi et al., J Immunol 2002 : In LPS stimulated MTECs, inhibition of c-Jun N-terminal kinase and p38 mitogen activated protein kinase significantly decreased RANTES, but did not affect MCP-1 mRNA induction
Bian et al., Exp Eye Res 2003 (Translocation, Genetic) : In addition, anti-inflammatory drugs dexamethasone ( DEX ) and cyclosporin A ( CSA ) both blocked activation of JNKS/SAPK and the cell-cell contact induced production of hRPE IL-8 and MCP-1 , while activation of p38 and ERK was only inhibited by DEX, but not by CSA
Vitale et al., J Immunol 2004 : We demonstrated that s-FKN abrogates the MCP-1 induced SAPK2/p38 activation as well as the upstream Pyk2 activity
Kato et al., J Am Soc Nephrol 2004 (Peritonitis) : mRNA expression of MCP-1 was abolished by NF-kappaB inhibition, but were not affected by the inhibition of ERK, JNK, or p38
Kim et al., Eur J Immunol 2005 (Bacteroides Infections...) : The p38 inhibitor SB203580 and the ERK inhibitor U0126 reduced not only AP-1 activity, but also decreased IL-8 and MCP-1 expression
Guest et al., Antioxid Redox Signal 2006 (Mechanotransduction, Cellular) : In conclusion, redox-sensitive activation of ERK1/2 ( MAPK ) and redox-insensitive activation of p38 ( MAPK ) regulate straininduced MCP-1 expression in RASM cells
Tsai et al., J Cell Biochem 2008 : Cilostazol significantly inhibited LPS induced activation of p38, JNK, and nuclear factor-kappaB, and the respective inhibitors of p38 and JNK greatly reduced the level of LPS induced MCP-1 and MMP-9, suggesting the involvement of the p38 and JNK pathways
Lee et al., J Clin Immunol 2009 : In addition, both p38 and ERK1/2 activation are essential for 30-kDa Ag-stimulated CCL2 production by monocytes
Frolova et al., Circ Res 2010 (Atherosclerosis...) : In addition, TSP-4 deficiency reduced endothelial cell activation ( expression of surface adhesion molecules ) and other markers of inflammation in the vascular wall ( decreased production of monocyte chemoattractant protein-1 and activation of p38 )
Li et al., J Clin Endocrinol Metab 2011 (Inflammation...) : In IL-1ß- or TNF-a stimulated first trimester decidual cells, NF?B inhibitor suppressed production of all six chemokines ; JUN NH2-terminal kinase inhibitor inhibited secretion of CCL2 , CCL4, and CCL5 ; and MAPK kinase and p38 inhibitor decreased production of CXCL8
Burke et al., PloS one 2012 : The IL-1ß mediated increase in CCL2 secretion was also impaired by p38 MAPK inhibition and by glucocorticoids