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MRXS5 — S100A12

Text-mined interactions from Literome

Ma et al., Neuroscience 2003 (Hyperalgesia...) : These findings are in contrast to our hypothesis, suggesting that peripherally over produced PGs following nerve injury and inflammation possibly contribute to the production of SP and CGRP in primary sensory neurons, to the up-regulation of dynorphin in the dorsal horn neurons, and finally to the mechanisms of neuropathic and inflammation pain
Taniguchi et al., Inflammopharmacology 2007 : This process is intervened by endogenous NO and CGRP in addition to prostaglandins (PGs), and the effect of PGs may be mediated by EP1 receptors